Literature DB >> 25828270

Impact of fibrate therapy on plasma plasminogen activator inhibitor-1: a systematic review and meta-analysis of randomized controlled trials.

Amirhossein Sahebkar1, Luis E Simental-Mendía2, Gerald F Watts3, Jonathan Golledge4.   

Abstract

OBJECTIVE: The aim of this systematic review was to perform a meta-analysis of randomized controlled trials (RCTs) examining the efficacy of fibrate therapy in reducing plasma concentration or activity of plasminogen activator inhibitor 1 (PAI-1).
METHODS: Scopus and MEDLINE databases were searched (up to October 15, 2014) to identify RCTs investigating whether fibrates lower plasma PAI-1 concentration or activity. A random-effects model and the generic inverse variance method were used for quantitative data synthesis. Sensitivity analyses were conducted using the one-study remove approach. Random-effects meta-regression was performed to assess the impact of potential moderators on the estimated effect sizes.
RESULTS: A total of 14 RCTs examining the effects of gemfibrozil (6 trials), bezafibrate (4 trials), and fenofibrate (5 trials) were included. Meta-analysis suggested that fibrate therapy did not significantly reduce plasma PAI-1 concentration (weighed mean difference [WMD]: -11.39 ng/mL, 95% CI: -26.64, 3.85, p=0.143) or activity (WMD: 2.02 U/mL, 95% CI: -0.87, 4.90, p=0.170). These results remained unchanged after subgroup analysis according to duration of treatment (<12 and ≥12 weeks) and type of fibrate administered (fenofibrate, bezafibrate or gemfibrozil). The estimated effects of fibrate therapy on plasma concentration and activity of PAI-1 were independent of treatment duration and changes in plasma triglyceride levels in the meta-regression analysis.
CONCLUSION: This meta-analysis of RCTs suggested that fibrate therapy does not reduce plasma concentration or activity of PAI-I. The putative benefits of fibrate therapy in patients with cardiovascular disease appear to be exerted via mechanisms independent of effects on PAI-1. Crown
Copyright © 2015. Published by Elsevier Ireland Ltd. All rights reserved.

Entities:  

Keywords:  Coronary heart disease; Fibric acid; Peroxisome proliferator-activated receptor; Plasminogen; Thrombosis

Mesh:

Substances:

Year:  2015        PMID: 25828270     DOI: 10.1016/j.atherosclerosis.2015.03.016

Source DB:  PubMed          Journal:  Atherosclerosis        ISSN: 0021-9150            Impact factor:   5.162


  3 in total

1.  Fibrates inhibit the apoptosis of Batten disease lymphoblast cells via autophagy recovery and regulation of mitochondrial membrane potential.

Authors:  Minho Hong; Ki Duk Song; Hak-Kyo Lee; SunShin Yi; Yong Seok Lee; Tae-Hwe Heo; Hyun Sik Jun; Sung-Jo Kim
Journal:  In Vitro Cell Dev Biol Anim       Date:  2015-12-10       Impact factor: 2.416

Review 2.  Hypofibrinolysis in diabetes: a therapeutic target for the reduction of cardiovascular risk.

Authors:  Katherine Kearney; Darren Tomlinson; Kerrie Smith; Ramzi Ajjan
Journal:  Cardiovasc Diabetol       Date:  2017-03-09       Impact factor: 9.951

3.  Effect of fenofibrate on plasma apolipoprotein C-III levels: a systematic review and meta-analysis of randomised placebo-controlled trials.

Authors:  Amirhossein Sahebkar; Luis E Simental-Mendía; Niki Katsiki; Željko Reiner; Maciej Banach; Matteo Pirro; Stephen L Atkin
Journal:  BMJ Open       Date:  2019-02-22       Impact factor: 2.692

  3 in total

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