Literature DB >> 25824144

Two Novel Homozygous Desmoplakin Mutations in Carvajal Syndrome.

Vered Molho-Pessach1,2, Sivan Sheffer1, Rula Siam1, Spiro Tams3, Ihab Siam1, Rula Awwad4, Sofia Babay2, Julius Golender5, Natalia Simanovsky6, Yuval Ramot1,2, Abraham Zlotogorski1,2.   

Abstract

BACKGROUND: Mutations in various desmosomal proteins were shown to cause inherited forms of cardiomyopathy. Carvajal syndrome (Online Mendelian Inheritance in Man [OMIM] 605676) is characterized by the association of dilated cardiomyopathy, striate palmoplantar keratoderma, and woolly hair. It is caused by homozygous as well as heterozygous mutations in DSP, which encodes the desmosomal plaque protein desmoplakin. An overlapping cardiocutaneous phenotype was also described with homozygous mutations in genes encoding two other desmosomal proteins; plakoglobin (Naxos disease; OMIM 601214) and desmocollin-2 (OMIM 610476).
METHODS: We performed clinical and molecular workups in two consanguineous Arab Palestinian families manifesting an autosomal recessive pattern of inheritance of the above mentioned clinical findings. Whole exome sequencing was employed in the search for the causing mutation.
RESULTS: Affected family members suffered from biventricular involvement and arrhythmogenic right ventricular dysplasia based on echocardiography and magnetic resonance imaging. One patient who underwent implantation of an implantable cardioverter-defibrillator (ICD) is still alive at the age of 59 years. Whole exome sequencing revealed two novel homozygous mutations in DSP, each affecting one family.
CONCLUSIONS: The association of woolly hair with palmoplantar keratoderma in a child should lead to a cardiac workup in the search for those at increased risk for sudden cardiac death. Early diagnosis and ICD implantation may be lifesaving. Whole exome sequencing should be utilized for rapid genetic analysis since the cardiocutaneous phenotype may result from mutations in one of several genes.
© 2015 Wiley Periodicals, Inc.

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Year:  2015        PMID: 25824144     DOI: 10.1111/pde.12541

Source DB:  PubMed          Journal:  Pediatr Dermatol        ISSN: 0736-8046            Impact factor:   1.588


  6 in total

1.  Autosomal dominant Carvajal plus syndrome due to the novel desmoplakin mutation c.1678A > T (p.Ile560Phe).

Authors:  Josef Finsterer; Claudia Stöllberger; Eva Wollmann; Susanne Dertinger; Franco Laccone
Journal:  Mol Genet Metab Rep       Date:  2016-06-02

Review 2.  Arrhythmogenic Right Ventricular Dysplasia in Neuromuscular Disorders.

Authors:  Josef Finsterer; Claudia Stöllberger
Journal:  Clin Med Insights Cardiol       Date:  2016-10-19

3.  A novel mutation in the desmoplakin gene in two female siblings with a rare form of dilated cardiomyopathy: Carvajal syndrome.

Authors:  Mehmet Gökhan Ramoğlu; Tayfun Uçar; Serdar Ceylaner; Semra Atalay; Ercan Tutar
Journal:  Anatol J Cardiol       Date:  2017-12       Impact factor: 1.596

4.  Dominant de novo DSP mutations cause erythrokeratodermia-cardiomyopathy syndrome.

Authors:  Lynn M Boyden; Chen Y Kam; Angela Hernández-Martín; Jing Zhou; Brittany G Craiglow; Robert Sidbury; Erin F Mathes; Sheilagh M Maguiness; Debra A Crumrine; Mary L Williams; Ronghua Hu; Richard P Lifton; Peter M Elias; Kathleen J Green; Keith A Choate
Journal:  Hum Mol Genet       Date:  2015-11-24       Impact factor: 6.150

Review 5.  Naxos disease: from the origin to today.

Authors:  Guo-Liang Li; Ardan M Saguner; Guy H Fontaine
Journal:  Orphanet J Rare Dis       Date:  2018-05-10       Impact factor: 4.123

6.  Desmoplakin maintains gap junctions by inhibiting Ras/MAPK and lysosomal degradation of connexin-43.

Authors:  Chen Yuan Kam; Adi D Dubash; Elisa Magistrati; Simona Polo; Karla J F Satchell; Farah Sheikh; Paul D Lampe; Kathleen J Green
Journal:  J Cell Biol       Date:  2018-06-29       Impact factor: 10.539

  6 in total

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