Literature DB >> 25775027

Correction: IFNγ signaling endows DCs with the capacity to control type I inflammation during parasitic infection through promoting T-bet+ regulatory T cells.

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Abstract

Entities:  

Year:  2015        PMID: 25775027      PMCID: PMC4361044          DOI: 10.1371/journal.ppat.1004741

Source DB:  PubMed          Journal:  PLoS Pathog        ISSN: 1553-7366            Impact factor:   6.823


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There are errors in the Funding section. The correct funding information is as follows: This work was supported by NIH grants AI089935 and AI108651 (L.-F.L). L.-F.L. is a Kimmel Scholar and a Hellman Fellow. H.-M.L is an Irvington Fellow of the Cancer Research Institute. The conditional IFNγR2 mutant mouse generation was supported by DFG (SFB 621 to W.M.) and MUGEN LSHG-CT-2005-005203. The initial functional characterization of IFNγR2 mutant mice was supported by the BBSRC PhD program at the University of Manchester. The aforementioned funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.
  1 in total

1.  IFNγ signaling endows DCs with the capacity to control type I inflammation during parasitic infection through promoting T-bet+ regulatory T cells.

Authors:  Hyang-Mi Lee; Anne Fleige; Ruth Forman; Sunglim Cho; Aly Azeem Khan; Ling-Li Lin; Duc T Nguyen; Aisling O'Hara-Hall; Zhinan Yin; Christopher A Hunter; Werner Muller; Li-Fan Lu
Journal:  PLoS Pathog       Date:  2015-02-06       Impact factor: 6.823

  1 in total

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