Literature DB >> 25768669

Iodide- and glucose-handling gene expression regulated by sorafenib or cabozantinib in papillary thyroid cancer.

Maomei Ruan1, Min Liu, Qianggang Dong, Libo Chen.   

Abstract

CONTEXT: The aberrant silencing of iodide-handling genes accompanied by up-regulation of glucose metabolism presents a major challenge for radioiodine treatment of papillary thyroid cancer (PTC).
OBJECTIVE: This study aimed to evaluate the effect of tyrosine kinase inhibitors on iodide-handling and glucose-handling gene expression in BHP 2-7 cells harboring RET/PTC1 rearrangement. MAIN OUTCOME MEASURES: In this in vitro study, the effects of sorafenib or cabozantinib on cell growth, cycles, and apoptosis were investigated by cell proliferation assay, cell cycle analysis, and Annexin V-FITC apoptosis assay, respectively. The effect of both agents on signal transduction pathways was evaluated using the Western blot. Quantitative real-time PCR, Western blot, immunofluorescence, and radioisotope uptake assays were used to assess iodide-handling and glucose-handling gene expression.
RESULTS: Both compounds inhibited cell proliferation in a time-dependent and dose-dependent manner and caused cell cycle arrest in the G0/G1 phase. Sorafenib blocked RET, AKT, and ERK1/2 phosphorylation, whereas cabozantinib blocked RET and AKT phosphorylation. The restoration of iodide-handling gene expression and inhibition of glucose transporter 1 and 3 expression could be induced by either drug. The robust expression of sodium/iodide symporter induced by either agent was confirmed, and (125)I uptake was correspondingly enhanced. (18)F-fluorodeoxyglucose accumulation was significantly decreased after treatment by either sorafenib or cabozantinib.
CONCLUSIONS: Sorafenib and cabozantinib had marked effects on cell proliferation, cell cycle arrest, and signal transduction pathways in PTC cells harboring RET/PTC1 rearrangement. Both agents could be potentially used to enhance the expression of iodide-handling genes and inhibit the expression of glucose transporter genes.

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Year:  2015        PMID: 25768669     DOI: 10.1210/jc.2014-3023

Source DB:  PubMed          Journal:  J Clin Endocrinol Metab        ISSN: 0021-972X            Impact factor:   5.958


  15 in total

Review 1.  Molecular mechanisms of radioactive iodine refractoriness in differentiated thyroid cancer: Impaired sodium iodide symporter (NIS) expression owing to altered signaling pathway activity and intracellular localization of NIS.

Authors:  Ji Min Oh; Byeong-Cheol Ahn
Journal:  Theranostics       Date:  2021-04-15       Impact factor: 11.556

2.  Predicting 131I-avidity of metastases from differentiated thyroid cancer using 18F-FDG PET/CT in postoperative patients with elevated thyroglobulin.

Authors:  Min Liu; Lingxiao Cheng; Yuchen Jin; Maomei Ruan; Shiwei Sheng; Libo Chen
Journal:  Sci Rep       Date:  2018-03-12       Impact factor: 4.379

3.  Reverting iodine avidity of radioactive-iodine refractory thyroid cancer with a new tyrosine kinase inhibitor (K905-0266) excavated by high-throughput NIS (sodium iodide symporter) enhancer screening platform using dual reporter gene system.

Authors:  Ji Min Oh; Senthilkumar Kalimuthu; Prakash Gangadaran; Se Hwan Baek; Liya Zhu; Ho Won Lee; Ramya Lakshmi Rajendran; Chae Moon Hong; Shin Young Jeong; Sang-Woo Lee; Jaetae Lee; Byeong-Cheol Ahn
Journal:  Oncotarget       Date:  2018-01-11

4.  HER inhibitor promotes BRAF/MEK inhibitor-induced redifferentiation in papillary thyroid cancer harboring BRAFV600E.

Authors:  Lingxiao Cheng; Yuchen Jin; Min Liu; Maomei Ruan; Libo Chen
Journal:  Oncotarget       Date:  2017-03-21

5.  Obatoclax and LY3009120 Efficiently Overcome Vemurafenib Resistance in Differentiated Thyroid Cancer.

Authors:  Wei-Jun Wei; Zhen-Kui Sun; Chen-Tian Shen; Hong-Jun Song; Xin-Yun Zhang; Zhong-Ling Qiu; Quan-Yong Luo
Journal:  Theranostics       Date:  2017-02-23       Impact factor: 11.556

6.  MAPK Inhibitors Enhance HDAC Inhibitor-Induced Redifferentiation in Papillary Thyroid Cancer Cells Harboring BRAF V600E: An In Vitro Study.

Authors:  Hao Fu; Lingxiao Cheng; Yuchen Jin; Lin Cheng; Min Liu; Libo Chen
Journal:  Mol Ther Oncolytics       Date:  2019-02-05       Impact factor: 7.200

7.  A novel strategy of transferring NIS protein to cells using extracellular vesicles leads to increase in iodine uptake and cytotoxicity.

Authors:  Seung Hyun Son; Prakash Gangadaran; Byeong-Cheol Ahn
Journal:  Int J Nanomedicine       Date:  2019-03-07

Review 8.  Mechanisms of regulating NIS transport to the cell membrane and redifferentiation therapy in thyroid cancer.

Authors:  X Cai; R Wang; J Tan; Z Meng; N Li
Journal:  Clin Transl Oncol       Date:  2021-06-08       Impact factor: 3.405

Review 9.  Molecular Targeted Therapies of Aggressive Thyroid Cancer.

Authors:  Silvia Martina Ferrari; Poupak Fallahi; Ugo Politti; Gabriele Materazzi; Enke Baldini; Salvatore Ulisse; Paolo Miccoli; Alessandro Antonelli
Journal:  Front Endocrinol (Lausanne)       Date:  2015-11-20       Impact factor: 5.555

10.  Combined tazemetostat and MAPKi enhances differentiation of papillary thyroid cancer cells harbouring BRAFV600E by synergistically decreasing global trimethylation of H3K27.

Authors:  Hao Fu; Lin Cheng; Ri Sa; Yuchen Jin; Libo Chen
Journal:  J Cell Mol Med       Date:  2020-01-22       Impact factor: 5.310

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