Literature DB >> 25759798

Novel NKX2-1 Frameshift Mutations in Patients with Atypical Phenotypes of the Brain-Lung-Thyroid Syndrome.

Tiziana de Filippis1, Federica Marelli1, Maria Cristina Vigone2, Marianna Di Frenna2, Giovanna Weber2, Luca Persani3.   

Abstract

OBJECTIVES: To verify the involvement of NKX2-1 gene in infants with brain-lung-thyroid (BLT) syndrome and hypothyroid phenotypes variable among congenital hypothyroidism (CH) or idiopathic mild hypothyroidism (IMH) of postnatal onset.
METHODS: The candidates were selected by a case-finding approach in 130 CH and 53 IMH infants. The NKX2-1 gene was analyzed by direct sequencing and multiplex ligation-dependent probe amplification. The variants were studied in vitro, by expression analyses and luciferase bioassay.
RESULTS: Four cases (3 CH and 1 IMH) consistent with BLT syndrome were identified. Two children were affected with respiratory distress and CH, but wild-type NKX2-1 gene. The remaining two presented choreic movements and no pulmonary involvement, but discrepant thyroid phenotypes: one had severe CH with lingual ectopy and the other one IMH with gland in situ. They were carriers of new de novo heterozygous frameshift mutations of NKX2-1 (c.177delG and c.153_166del14). The c.177delG leads to a prematurely truncated protein (p.H60TfsX11) with undetectable activity in vitro. The c.153_166del14 leads to the generation of an elongated aberrant protein (p.A52RfsX351) able to translocate into the nucleus, but completely inactive on a responsive promoter.
CONCLUSIONS: Two novel heterozygous frameshift mutations of NKX2-1 were identified in 2 cases selected on the basis of a BLT-like phenotype among 183 hypothyroid infants. The atypical hypothyroid phenotypes of these 2 children (CH with lingual ectopy or IMH of postnatal onset) further expand the clinical spectrum that can be associated with NKX2-1 mutations.

Entities:  

Keywords:  Brain-lung-thyroid syndrome; Choreoathetosis; Congenital hypothyroidism; NKX2-1; Thyroid dysgenesis; Thyroid ectopy

Year:  2014        PMID: 25759798      PMCID: PMC4311306          DOI: 10.1159/000366274

Source DB:  PubMed          Journal:  Eur Thyroid J        ISSN: 2235-0640


  19 in total

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5.  NKX2-1 mutations leading to surfactant protein promoter dysregulation cause interstitial lung disease in "Brain-Lung-Thyroid Syndrome".

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8.  Mutations in TITF-1 are associated with benign hereditary chorea.

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9.  A 7-year experience with low blood TSH cutoff levels for neonatal screening reveals an unsuspected frequency of congenital hypothyroidism (CH).

Authors:  Carlo Corbetta; Giovanna Weber; Francesca Cortinovis; Davide Calebiro; Arianna Passoni; Maria C Vigone; Paolo Beck-Peccoz; Giuseppe Chiumello; Luca Persani
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10.  European Society for Paediatric Endocrinology consensus guidelines on screening, diagnosis, and management of congenital hypothyroidism.

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Journal:  J Clin Endocrinol Metab       Date:  2014-01-21       Impact factor: 5.958

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