Literature DB >> 25752761

A complicated case of atypical hemolytic uremic syndrome with frequent relapses under eculizumab.

Gesa Schalk1, Michael Kirschfink, Cyrill Wehling, Sara Gastoldi, Carsten Bergmann, Bernd Hoppe, Lutz T Weber.   

Abstract

BACKGROUND: Atypical hemolytic uremic syndrome (aHUS) is a form of thrombotic microangiopathy characterized by uncontrolled activation of the alternative complement pathway with consecutive generation of the terminal complement complex. Mortality is increased, particularly in the first year of the disease. Therapeutic options include plasma therapy and terminal complement blockade using the anti-C5 monoclonal antibody eculizumab. Eculizumab prevents activation of the terminal sequence of the complement cascade and formation of the potentially lytic terminal complement complex (C5b-9). CASE-DIAGNOSIS/TREATMENT: We report a 3-year-old boy with aHUS due to a novel heterozygous truncating complement Factor H mutation in combination with other changes known to be associated with an increased risk for aHUS. Despite eculizumab treatment and maximal suppression of the classical and alternative complement pathways, C3d and sC5b-9 remained consistently elevated and the patient showed repeated relapses.
CONCLUSIONS: Not every patient with aHUS and uncontrolled complement activation shows optimal therapeutic response to eculizumab with the recommended or even increased dosing regimen. Reliable outcome measures to determine the efficacy of treatment have to be defined.

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Year:  2015        PMID: 25752761     DOI: 10.1007/s00467-015-3078-6

Source DB:  PubMed          Journal:  Pediatr Nephrol        ISSN: 0931-041X            Impact factor:   3.714


  14 in total

1.  Tailored eculizumab regimen for patients with atypical hemolytic uremic syndrome: requirement for comprehensive complement analysis.

Authors:  C Wehling; M Kirschfink
Journal:  J Thromb Haemost       Date:  2014-07-15       Impact factor: 5.824

Review 2.  Use of eculizumab for atypical haemolytic uraemic syndrome and C3 glomerulopathies.

Authors:  Julien Zuber; Fadi Fakhouri; Lubka T Roumenina; Chantal Loirat; Véronique Frémeaux-Bacchi
Journal:  Nat Rev Nephrol       Date:  2012-10-02       Impact factor: 28.314

3.  Blockade of C5a and C5b-9 generation inhibits leukocyte and platelet activation during extracorporeal circulation.

Authors:  C S Rinder; H M Rinder; B R Smith; J C Fitch; M J Smith; J B Tracey; L A Matis; S P Squinto; S A Rollins
Journal:  J Clin Invest       Date:  1995-09       Impact factor: 14.808

4.  Mutations in components of complement influence the outcome of Factor I-associated atypical hemolytic uremic syndrome.

Authors:  Frank Bienaime; Marie-Agnes Dragon-Durey; Catherine H Regnier; Sara C Nilsson; Wing H Kwan; Jacques Blouin; Mathieu Jablonski; Nicolas Renault; Marie-Anne Rameix-Welti; Chantal Loirat; Catherine Sautés-Fridman; Bruno O Villoutreix; Anna M Blom; Veronique Fremeaux-Bacchi
Journal:  Kidney Int       Date:  2009-12-16       Impact factor: 10.612

Review 5.  Genetics and complement in atypical HUS.

Authors:  David Kavanagh; Tim Goodship
Journal:  Pediatr Nephrol       Date:  2010-06-06       Impact factor: 3.714

6.  Dynamics of complement activation in aHUS and how to monitor eculizumab therapy.

Authors:  Marina Noris; Miriam Galbusera; Sara Gastoldi; Paolo Macor; Federica Banterla; Elena Bresin; Claudio Tripodo; Serena Bettoni; Roberta Donadelli; Elisabetta Valoti; Francesco Tedesco; Alessandro Amore; Rosanna Coppo; Piero Ruggenenti; Eliana Gotti; Giuseppe Remuzzi
Journal:  Blood       Date:  2014-07-18       Impact factor: 22.113

7.  Association of factor H autoantibodies with deletions of CFHR1, CFHR3, CFHR4, and with mutations in CFH, CFI, CD46, and C3 in patients with atypical hemolytic uremic syndrome.

Authors:  Iain Moore; Lisa Strain; Isabel Pappworth; David Kavanagh; Paul N Barlow; Andrew P Herbert; Christoph Q Schmidt; Scott J Staniforth; Lucy V Holmes; Roy Ward; Lynn Morgan; Timothy H J Goodship; Kevin J Marchbank
Journal:  Blood       Date:  2009-10-27       Impact factor: 22.113

Review 8.  STEC-HUS, atypical HUS and TTP are all diseases of complement activation.

Authors:  Marina Noris; Federica Mescia; Giuseppe Remuzzi
Journal:  Nat Rev Nephrol       Date:  2012-09-18       Impact factor: 28.314

9.  Terminal complement inhibitor eculizumab in atypical hemolytic-uremic syndrome.

Authors:  C M Legendre; C Licht; P Muus; L A Greenbaum; S Babu; C Bedrosian; C Bingham; D J Cohen; Y Delmas; K Douglas; F Eitner; T Feldkamp; D Fouque; R R Furman; O Gaber; M Herthelius; M Hourmant; D Karpman; Y Lebranchu; C Mariat; J Menne; B Moulin; J Nürnberger; M Ogawa; G Remuzzi; T Richard; R Sberro-Soussan; B Severino; N S Sheerin; A Trivelli; L B Zimmerhackl; T Goodship; C Loirat
Journal:  N Engl J Med       Date:  2013-06-06       Impact factor: 91.245

10.  Long-term eculizumab improves clinical outcomes in atypical hemolytic uremic syndrome.

Authors:  Ramon Vilalta; Enrique Lara; Alvaro Madrid; Sara Chocron; Marina Muñoz; Alex Casquero; Jose Nieto
Journal:  Pediatr Nephrol       Date:  2012-08-14       Impact factor: 3.714

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  10 in total

1.  Thrombotic microangiopathy caused by methionine synthase deficiency: diagnosis and treatment pitfalls.

Authors:  Maria Helena Vaisbich; Andressa Braga; Maria Gabrielle; Clarissa Bueno; Flávia Piazzon; Fernando Kok
Journal:  Pediatr Nephrol       Date:  2017-02-16       Impact factor: 3.714

2.  Incomplete inhibition by eculizumab: mechanistic evidence for residual C5 activity during strong complement activation.

Authors:  Markus J Harder; Nadine Kuhn; Hubert Schrezenmeier; Britta Höchsmann; Inge von Zabern; Christof Weinstock; Thomas Simmet; Daniel Ricklin; John D Lambris; Arne Skerra; Markus Anliker; Christoph Q Schmidt
Journal:  Blood       Date:  2016-12-27       Impact factor: 22.113

3.  Monitoring of complement activation biomarkers and eculizumab in complement-mediated renal disorders.

Authors:  C Wehling; O Amon; M Bommer; B Hoppe; K Kentouche; G Schalk; R Weimer; M Wiesener; B Hohenstein; B Tönshoff; R Büscher; H Fehrenbach; Ö-N Gök; M Kirschfink
Journal:  Clin Exp Immunol       Date:  2016-11-25       Impact factor: 4.330

4.  The MFHR1 Fusion Protein Is a Novel Synthetic Multitarget Complement Inhibitor with Therapeutic Potential.

Authors:  Stefan Michelfelder; Friedericke Fischer; Astrid Wäldin; Kim V Hörle; Martin Pohl; Juliana Parsons; Ralf Reski; Eva L Decker; Peter F Zipfel; Christine Skerka; Karsten Häffner
Journal:  J Am Soc Nephrol       Date:  2018-01-15       Impact factor: 10.121

5.  Liver transplantation for aHUS: still needed in the eculizumab era?

Authors:  Rosanna Coppo; Roberto Bonaudo; R Licia Peruzzi; Alessandro Amore; Andrea Brunati; Renato Romagnoli; Mauro Salizzoni; Miriam Galbusera; Eliana Gotti; Erica Daina; Marina Noris; Giuseppe Remuzzi
Journal:  Pediatr Nephrol       Date:  2015-11-24       Impact factor: 3.714

6.  Adjustment of Eculizumab Dosage Pattern in Patients with Atypical Hemolytic Uremic Syndrome with Suboptimal Response to Standard Treatment Pattern.

Authors:  Camino García Monteavaro; Carmen Peralta Roselló; Borja Quiroga; José María Baltar Martín; Lorena Castillo Eraso; Fernando de Álvaro Moreno; Alberto Martínez Vea; María Teresa Visus-Fernández de Manzanos
Journal:  Case Rep Nephrol       Date:  2016-11-29

7.  Current evidence on the discontinuation of eculizumab in patients with atypical haemolytic uraemic syndrome.

Authors:  Manuel Macia; Fernando de Alvaro Moreno; Tina Dutt; Ingela Fehrman; Karine Hadaya; Christoph Gasteyger; Nils Heyne
Journal:  Clin Kidney J       Date:  2016-12-22

Review 8.  Critical appraisal of eculizumab for atypical hemolytic uremic syndrome.

Authors:  Lilian M Pereira Palma; Craig B Langman
Journal:  J Blood Med       Date:  2016-04-12

9.  Kidney Transplantation in Patients with Atypical Hemolytic Uremic Syndrome due to Complement Factor H Deficiency: Impact of Liver Transplantation.

Authors:  Sejin Kim; Eujin Park; Sang Il Min; Nam Joon Yi; Jongwon Ha; Il Soo Ha; Hae Il Cheong; Hee Gyung Kang
Journal:  J Korean Med Sci       Date:  2018-01-01       Impact factor: 2.153

10.  Properdin Is a Key Player in Lysis of Red Blood Cells and Complement Activation on Endothelial Cells in Hemolytic Anemias Caused by Complement Dysregulation.

Authors:  Jin Y Chen; Neeti S Galwankar; Heather N Emch; Smrithi S Menon; Claudio Cortes; Joshua M Thurman; Samuel A Merrill; Robert A Brodsky; Viviana P Ferreira
Journal:  Front Immunol       Date:  2020-07-22       Impact factor: 7.561

  10 in total

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