Literature DB >> 25736794

TRP Channels Localize to Subdomains of the Apical Plasma Membrane in Human Fetal Retinal Pigment Epithelium.

Peter Y Zhao1, Geliang Gan1, Shaomin Peng1, Shao-Bin Wang2, Bo Chen3, Ron A Adelman3, Lawrence J Rizzolo2.   

Abstract

PURPOSE: Calcium regulates many functions of the RPE. Its concentration in the subretinal space and RPE cytoplasm is closely regulated. Transient receptor potential (TRP) channels are a superfamily of ion channels that are moderately calcium-selective. This study investigates the subcellular localization and potential functions of TRP channels in a first-passage culture model of human fetal RPE (hfRPE).
METHODS: The RPE isolated from 15- to 16-week gestation fetuses were maintained in serum-free media. Cultures were treated with barium chloride (BaCl2) in the absence and presence of TRP channel inhibitors and monitored by the transepithelial electrical resistance (TER). The expression of TRP channels was determined using quantitative RT-PCR, immunoblotting, and immunofluorescence confocal microscopy.
RESULTS: Barium chloride substantially decreased TER and disrupted cell-cell contacts when added to the apical surface of RPE, but not when added to the basolateral surface. The effect could be partially blocked by the general TRP inhibitor, lanthanum chloride (LaCl3, ~75%), or an inhibitor of calpain (~25%). Family member-specific inhibitors, ML204 (TRPC4) and HC-067047 (TRPV4), had no effect on basal channel activity. Expression of TRPC4, TRPM1, TRPM3, TRPM7, and TRPV4 was detected by RT-PCR and immunoblotting. The TRPM3 localized to the base of the primary cilium, and TRPC4 and TRPM3 localized to apical tight junctions. The TRPV4 localized to apical microvilli in a small subset of cells.
CONCLUSIONS: The TRP channels localized to subdomains of the apical membrane, and BaCl2 was only able to dissociate tight junctions when presented to the apical membrane. The data suggest a potential role for TRP channels as sensors of [Ca(2+)] in the subretinal space. Copyright 2015 The Association for Research in Vision and Ophthalmology, Inc.

Entities:  

Keywords:  TRP channels; retinal pigment epithelium; subretinal space; tight junction

Mesh:

Substances:

Year:  2015        PMID: 25736794      PMCID: PMC4364639          DOI: 10.1167/iovs.14-15738

Source DB:  PubMed          Journal:  Invest Ophthalmol Vis Sci        ISSN: 0146-0404            Impact factor:   4.799


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2.  TRPM7 regulates cell adhesion by controlling the calcium-dependent protease calpain.

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Review 3.  Permeation and selectivity of TRP channels.

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4.  Confluent monolayers of cultured human fetal retinal pigment epithelium exhibit morphology and physiology of native tissue.

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5.  A novel serum-free method for culturing human prenatal retinal pigment epithelial cells.

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2.  Roles of transient receptor potential channels in regulation of vascular and epithelial barriers.

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3.  Primary cilia regulate the osmotic stress response of renal epithelial cells through TRPM3.

Authors:  Brian J Siroky; Nancy K Kleene; Steven J Kleene; Charles D Varnell; Raven G Comer; Jialiu Liu; Lu Lu; Nolan W Pachciarz; John J Bissler; Bradley P Dixon
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4.  Cerebrospinal Fluid-Contacting Neurons Sense pH Changes and Motion in the Hypothalamus.

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7.  TRPV4 inhibition prevents increased water diffusion and blood-retina barrier breakdown in the retina of streptozotocin-induced diabetic mice.

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Review 8.  Diseases associated with leaky hemichannels.

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9.  Early Functional and Morphologic Abnormalities in the Diabetic Nyxnob Mouse Retina.

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10.  Dual contribution of TRPV4 antagonism in the regulatory effect of vasoinhibins on blood-retinal barrier permeability: diabetic milieu makes a difference.

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