| Literature DB >> 25696734 |
B J J M Brundel, R H Henning, W H van Gilst, I C van Gelder, H J G M Crijns.
Abstract
The chance that the treatment of atrial fibrillation (AF) is successful depends on the duration of the arrhythmia. The low efficacy of cardioversion therapy after long-term AF can be explained by the occurrence of cellular adaptation mechanisms. In this article we describe ion-channel protein remodelling and structural changes in the atria of patients with persistent and paroxysmal AF and its relation with electrical remodelling.Entities:
Keywords: atrial fibrillation; calpain; human; ion channels; remodelling
Year: 2001 PMID: 25696734 PMCID: PMC2499617
Source DB: PubMed Journal: Neth Heart J ISSN: 1568-5888 Impact factor: 2.380