Literature DB >> 2569472

Clonal loss of one chromosome 11 in a parathyroid adenoma.

A Arnold1, H G Kim.   

Abstract

Traditional cytogenetic approaches have been unsuccessful in the study of parathyroid adenomas. We now describe one parathyroid adenoma in which a molecular cytogenetic approach revealed clonal loss of one chromosome 11. Restriction fragment length polymorphism analysis of the patient's normal leukocyte DNA demonstrated heterozygosity at four loci (PTH, INT2, APOA1, and ETS1) that span the length of chromosome 11. However, the adenoma DNA showed clonal deletion of one allele, i.e. loss of heterozygosity, at each locus. Use of five nonpolymorphic probes from chromosome 11 was consistent with 50% loss of total chromosome 11 DNA in the adenoma. No tumor-specific loss of heterozygosity was observed when restriction fragment length polymorphisms from five other autosomes (no. 1, 5, 6, 7, and 12) were analyzed, and an X-chromosome probe also showed no tumor DNA loss. We have demonstrated tumor-specific chromosome loss in a parathyroid adenoma; such a lesion has been described only rarely in benign tumors. Our finding adds to the evidence for monoclonality in parathyroid adenomatosis, indicates that only one PTH gene copy is sufficient for hyperparathyroid tumor function, and raises the possibility that a tumor-suppressor gene important in the development of nonfamilial parathyroid neoplasia is located on chromosome 11.

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Year:  1989        PMID: 2569472     DOI: 10.1210/jcem-69-3-496

Source DB:  PubMed          Journal:  J Clin Endocrinol Metab        ISSN: 0021-972X            Impact factor:   5.958


  8 in total

1.  Clonality in Thyroid Nodules: The Hyperplasia-Neoplasia Sequence.

Authors:  Zubair W. Baloch; Virginia A. LiVolsi
Journal:  Endocr Pathol       Date:  1998       Impact factor: 3.943

2.  Controversies and advances in primary hyperparathyroidism.

Authors:  J A Norton
Journal:  Ann Surg       Date:  1992-04       Impact factor: 12.969

3.  Expression and somatic mutations of SDHAF2 (SDH5), a novel endocrine tumor suppressor gene in parathyroid tumors of primary hyperparathyroidism.

Authors:  Lee F Starker; Alberto Delgado-Verdugo; Robert Udelsman; Peyman Björklund; Tobias Carling
Journal:  Endocrine       Date:  2010-10-23       Impact factor: 3.633

Review 4.  "Asymptomatic" and symptomatic primary hyperparathyroidism.

Authors:  J A Fischer
Journal:  Clin Investig       Date:  1993-07

Review 5.  Expression of p53, Ki-67 and Bcl-2 in parathyroid adenoma and residual normal tissue.

Authors:  Tuvia Hadar; Jacob Shvero; Eitan Yaniv; Eduard Ram; Itzhak Shvili; Rumelia Koren
Journal:  Pathol Oncol Res       Date:  2005-03-31       Impact factor: 3.201

6.  Clonal analysis of solitary follicular nodules in the thyroid.

Authors:  D G Hicks; V A LiVolsi; J A Neidich; J M Puck; J A Kant
Journal:  Am J Pathol       Date:  1990-09       Impact factor: 4.307

7.  Evidence of a stabilizing mutation of β-catenin encoded by CTNNB1 exon 3 in a large series of sporadic parathyroid adenomas.

Authors:  Lee F Starker; Annabelle L Fonseca; Annabelle Fonseca; Göran Akerström; Peyman Björklund; Gunnar Westin; Tobias Carling
Journal:  Endocrine       Date:  2012-05-11       Impact factor: 3.633

8.  The DNA methylome of benign and malignant parathyroid tumors.

Authors:  Lee F Starker; Jessica Svedlund; Robert Udelsman; Henning Dralle; Göran Akerström; Gunnar Westin; Richard P Lifton; Peyman Björklund; Tobias Carling
Journal:  Genes Chromosomes Cancer       Date:  2011-06-02       Impact factor: 5.006

  8 in total

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