Literature DB >> 25645639

Further evidence of the association between LQT syndrome and epilepsy in a family with KCNQ1 pathogenic variant.

Coloma Tiron1, Coloma Tiron de Llano, Oscar Campuzano2, Alexandra Pérez-Serra3, Irene Mademont3, Monica Coll3, Catarina Allegue3, Anna Iglesias3, Sara Partemi4, Pasquale Striano5, Antonio Oliva4, Ramon Brugada6.   

Abstract

PURPOSE: Ion channels are expressed both in the heart and in the brain, being advocated as responsible for sudden unexpected death in epilepsy but few pathogenic mutations have been identified. We aim to identify a novel gen associated with channelopathies and epilepsy in a family.
METHODS: We assessed a family showing epilepsy concomitant with LQTS. Index case showed prolonged QT interval. His father suffers of LQT and epilepsy. We performed a direct sequencing analysis of KCNQ1, KCNH2, KCNE1, KCNE2 and SCN5A genes.
RESULTS: We identified a non-synonymous heterozygous missense pathogenic mutation (p.L273F) in exon 6 of the KCNQ1 gene. All clinically affected relatives carried the same mutation.
CONCLUSION: We report, for a first time, a KCNQ1 mutation in a family suffering of both phenotypes, suggesting that KCNQ1 genetic variations may confer susceptibility for recurrent seizure activity increasing the risk or lead to sudden death.
Copyright © 2015 British Epilepsy Association. Published by Elsevier Ltd. All rights reserved.

Entities:  

Keywords:  Epilepsy; KCNQ1; Long QT syndrome

Mesh:

Substances:

Year:  2015        PMID: 25645639     DOI: 10.1016/j.seizure.2015.01.003

Source DB:  PubMed          Journal:  Seizure        ISSN: 1059-1311            Impact factor:   3.184


  22 in total

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