Literature DB >> 25624412

Bayesian analyses demonstrate tissue blood volume is not decreased during acute sickle cell pain episodes: A preliminary study.

Maria Perry1, Jena Simon2, Daniel Gareau3, Jeffrey Glassberg4.   

Abstract

BACKGROUND: Pain is the most common complication of Sickle Cell Disease (SCD). Tissue oximetry properties in SCD during steady state and acute pain are not well described.
METHODS: This was a cross sectional study of tissue oximetry properties in individuals with SCD during steady state, acute pain and healthy controls without SCD. A novel tissue oximetry device was used to better account for tissue pigmentation interference. We hypothesized that during acute SCD pain, blood volume to painful areas would be at least 10% less than steady state. Bayesian analyses of the data (with flat piors) were planned a priori because of the small projected sample size.
RESULTS: The sample included 14 individuals (4 during crisis, 5 steady state, and 5 controls). In individuals with SCD, blood volume to the lower back was higher during crisis (0.18% of tissue volume vs. 0.14% ). Bayesian analyses yielded a 3% probability that our hypothesis (that blood volume would decrease by 10% ) was correct.
CONCLUSIONS: During acute SCD pain, blood volume to painful areas is not decreased. Bayesian analyses were useful for interpretation of small sample data and may have utility in early phase trials for rare diseases.

Entities:  

Keywords:  Sickle cell disease; pain crisis; tissue oximetry

Mesh:

Year:  2016        PMID: 25624412      PMCID: PMC4515395          DOI: 10.3233/CH-141927

Source DB:  PubMed          Journal:  Clin Hemorheol Microcirc        ISSN: 1386-0291            Impact factor:   2.375


  27 in total

Review 1.  Sickle cell vasoocclusion: heterotypic, multicellular aggregations driven by leukocyte adhesion.

Authors:  Paul S Frenette
Journal:  Microcirculation       Date:  2004-03       Impact factor: 2.628

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Journal:  N Engl J Med       Date:  1984-12-13       Impact factor: 91.245

5.  Sickle cell adhesion depends on hemodynamics and endothelial activation.

Authors:  Matthew C Wagner; James R Eckman; Timothy M Wick
Journal:  J Lab Clin Med       Date:  2004-11

6.  Assessment of cerebral tissue oxygenation in patients with sickle cell disease: effect of transfusion therapy.

Authors:  Ashok Raj; Salvatore J Bertolone; Sara Mangold; Harvey L Edmonds
Journal:  J Pediatr Hematol Oncol       Date:  2004-05       Impact factor: 1.289

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Journal:  J Clin Pathol       Date:  1992-10       Impact factor: 3.411

8.  The painful crisis of homozygous sickle cell disease: clinical features.

Authors:  G R Serjeant; C D Ceulaer; R Lethbridge; J Morris; A Singhal; P W Thomas
Journal:  Br J Haematol       Date:  1994-07       Impact factor: 6.998

9.  Imaging hemoglobin oxygen saturation in sickle cell disease patients using noninvasive visible reflectance hyperspectral techniques: effects of nitric oxide.

Authors:  Karel J Zuzak; Mark T Gladwin; Richard O Cannon; Ira W Levin
Journal:  Am J Physiol Heart Circ Physiol       Date:  2003-06-05       Impact factor: 4.733

10.  Continuous, noninvasive, and localized microvascular tissue oximetry using visible light spectroscopy.

Authors:  David A Benaron; Ilian H Parachikov; Shai Friedland; Roy Soetikno; John Brock-Utne; Peter J A van der Starre; Camran Nezhat; Martha K Terris; Peter G Maxim; Jeffrey J L Carson; Mahmood K Razavi; Hayes B Gladstone; Edgar F Fincher; Christopher P Hsu; F Landon Clark; Wai-Fung Cheong; Joshua L Duckworth; David K Stevenson
Journal:  Anesthesiology       Date:  2004-06       Impact factor: 7.892

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