Literature DB >> 25623250

Cell-specific effects in different immune subsets associated with SOCS1 genotypes in multiple sclerosis.

Aitzkoa Lopez de Lapuente1, María Jesús Pinto-Medel2, Ianire Astobiza1, Iraide Alloza3, Manuel Comabella4, Sunny Malhotra4, Xavier Montalban4, Uwe K Zettl5, Alfredo Rodríguez-Antigüedad6, Oscar Fernández2, Koen Vandenbroeck7.   

Abstract

BACKGROUND: Single nucleotide polymorphisms (SNPs) near SOCS1 are associated with multiple sclerosis (MS), but the most important SNPs in the area and mechanisms by which they influence the disease are unknown.
METHODS: A haplotype-tagging association study was performed covering 60.5kbp around SOCS1, and the index SNP was validated in a total of 2292 individuals. mRNA expression of SOCS1 and nearby genes was measured in MS patients with different disease courses and healthy controls. SOCS1 protein expression was studied by flow cytometry in a separate cohort of patients and controls. Differentiation and maturation of monocyte-derived dendritic cells (moDCs) were also studied.
RESULTS: One SNP, rs423674, reached genome-wide significance. No genotype-specific mRNA expression differences were seen, but, by flow cytometry, significant interactions were observed between genotypes for rs423674 and disease activity (relapse or remission) in B cells and regulatory T cells. Furthermore, homozygotes for the risk allele (GG) showed higher levels of CD1a and CD86 than carriers of the protective allele (GT) in immature moDCs and a greater increase of HLA-DR+ cell percentage than GT heterozygotes upon maturation.
CONCLUSIONS: rs423674, or its genetic proxies, may influence MS risk by modulating SOCS1 expression in a cell-specific manner and by influencing dendritic cell function.
© The Author(s), 2015.

Entities:  

Keywords:  SNP; SOCS1; expression; multiple sclerosis; relapsing–remitting

Mesh:

Substances:

Year:  2015        PMID: 25623250     DOI: 10.1177/1352458514566418

Source DB:  PubMed          Journal:  Mult Scler        ISSN: 1352-4585            Impact factor:   6.312


  5 in total

Review 1.  Negative Regulation of Cytokine Signaling in Immunity.

Authors:  Akihiko Yoshimura; Minako Ito; Shunsuke Chikuma; Takashi Akanuma; Hiroko Nakatsukasa
Journal:  Cold Spring Harb Perspect Biol       Date:  2018-07-02       Impact factor: 10.005

Review 2.  Opportunities for Translation from the Bench: Therapeutic Intervention of the JAK/STAT Pathway in Neuroinflammatory Diseases.

Authors:  Yudong Liu; Sara A Gibson; Etty N Benveniste; Hongwei Qin
Journal:  Crit Rev Immunol       Date:  2015       Impact factor: 2.214

3.  Multiple Sclerosis Risk Allele in CLEC16A Acts as an Expression Quantitative Trait Locus for CLEC16A and SOCS1 in CD4+ T Cells.

Authors:  Ingvild S Leikfoss; Pankaj K Keshari; Marte W Gustavsen; Anja Bjølgerud; Ina S Brorson; Elisabeth G Celius; Anne Spurkland; Steffan D Bos; Hanne F Harbo; Tone Berge
Journal:  PLoS One       Date:  2015-07-23       Impact factor: 3.240

4.  Suppressor of cytokine signaling-1 mimetic peptides attenuate lymphocyte activation in the MRL/lpr mouse autoimmune model.

Authors:  Jatin Sharma; Teresa D Collins; Tracoyia Roach; Shiwangi Mishra; Brandon K Lam; Zaynab Sidi Mohamed; Antia E Veal; Timothy B Polk; Amari Jones; Caleb Cornaby; Mohammed I Haider; Leilani Zeumer-Spataro; Howard M Johnson; Laurence M Morel; Joseph Larkin
Journal:  Sci Rep       Date:  2021-03-18       Impact factor: 4.996

Review 5.  Regulation of Macrophage, Dendritic Cell, and Microglial Phenotype and Function by the SOCS Proteins.

Authors:  Sarah M McCormick; Nicola M Heller
Journal:  Front Immunol       Date:  2015-10-27       Impact factor: 7.561

  5 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.