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15 in total

1. GPR40 receptor agonist TAK-875 improves cognitive deficits and reduces β-amyloid production in APPswe/PS1dE9 mice.

Authors: Chao Liu; Zhao-Yan Cheng; Qing-Peng Xia; Yu-Hui Hu; Chen Wang; Ling He
Journal: Psychopharmacology (Berl) Date: 2021-06-26 Impact factor: 4.530

2. Development of AD-Like Pathology in Skeletal Muscle.

Authors: X Chen; N M Miller; Z Afghah; J D Geiger
Journal: J Parkinsons Dis Alzheimers Dis Date: 2019-04-02

Review 3. Interactions of pathological proteins in neurodegenerative diseases.

Authors: Tara L Spires-Jones; Johannes Attems; Dietmar Rudolf Thal
Journal: Acta Neuropathol Date: 2017-04-11 Impact factor: 17.088

4. Tau Pathology Promotes the Reorganization of the Extracellular Matrix and Inhibits the Formation of Perineuronal Nets by Regulating the Expression and the Distribution of Hyaluronic Acid Synthases.

Authors: Yin Li; Ze-Xu Li; Tan Jin; Zhan-You Wang; Pu Zhao
Journal: J Alzheimers Dis Date: 2017 Impact factor: 4.472

5. EGCG Nanoparticles Attenuate Aluminum Chloride Induced Neurobehavioral Deficits, Beta Amyloid and Tau Pathology in a Rat Model of Alzheimer's Disease.

Authors: Neha Atulkumar Singh; Vaishali Bhardwaj; Chandrika Ravi; Nithya Ramesh; Abul Kalam Azad Mandal; Zaved Ahmed Khan
Journal: Front Aging Neurosci Date: 2018-08-13 Impact factor: 5.750

10. Tryptophan-galactosylamine conjugates inhibit and disaggregate amyloid fibrils of Aβ42 and hIAPP peptides while reducing their toxicity.

Authors: Ashim Paul; Moran Frenkel-Pinter; Daniela Escobar Alvarez; Giulia Milordini; Ehud Gazit; Elsa Zacco; Daniel Segal
Journal: Commun Biol Date: 2020-09-02

Protein aggregation in Alzheimer's disease: Aβ and τ and their potential roles in the pathogenesis of AD.

1. GPR40 receptor agonist TAK-875 improves cognitive deficits and reduces β-amyloid production in APPswe/PS1dE9 mice.

2. Development of AD-Like Pathology in Skeletal Muscle.

Review 3. Interactions of pathological proteins in neurodegenerative diseases.

4. Tau Pathology Promotes the Reorganization of the Extracellular Matrix and Inhibits the Formation of Perineuronal Nets by Regulating the Expression and the Distribution of Hyaluronic Acid Synthases.

5. EGCG Nanoparticles Attenuate Aluminum Chloride Induced Neurobehavioral Deficits, Beta Amyloid and Tau Pathology in a Rat Model of Alzheimer's Disease.

6. Defining and predicting transdiagnostic categories of neurodegenerative disease.

Review 7. Biomaterials for the Treatment of Alzheimer's Disease.

8. Cognitive function in very old men does not correlate to biomarkers of Alzheimer's disease.

9. Different Amyloid-β Self-Assemblies Have Distinct Effects on Intracellular Tau Aggregation.

10. Tryptophan-galactosylamine conjugates inhibit and disaggregate amyloid fibrils of Aβ42 and hIAPP peptides while reducing their toxicity.