| Literature DB >> 25549669 |
Juliane Proft1, Norbert Weiss2.
Abstract
Neuronal voltage-gated calcium channels have evolved as one of the most important players for calcium entry into presynaptic endings responsible for the release of neurotransmitters. In turn, and to fine-tune synaptic activity and neuronal communication, numerous neurotransmitters exert a potent negative feedback over the calcium signal provided by G protein-coupled receptors. This regulation pathway of physiologic importance is also extensively exploited for therapeutic purposes, for instance in the treatment of neuropathic pain by morphine and other μ-opioid receptor agonists. However, despite more than three decades of intensive research, important questions remain unsolved regarding the molecular and cellular mechanisms of direct G protein inhibition of voltage-gated calcium channels. In this study, we revisit this particular regulation and explore new considerations.Entities:
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Year: 2014 PMID: 25549669 DOI: 10.1124/mol.114.096008
Source DB: PubMed Journal: Mol Pharmacol ISSN: 0026-895X Impact factor: 4.436