Literature DB >> 25515581

A new small molecule for treating inflammation and chorioretinal neovascularization in relapsing-remitting and chronic experimental autoimmune uveitis.

Maria Diedrichs-Möhring1, Johann Leban2, Stefan Strobl2, Franz Obermayr3, Gerhild Wildner1.   

Abstract

PURPOSE: We investigated the effect of PP-001, a new small molecule inhibitor of dihydro-orotate dehydrogenase in two experimental rat experimental autoimmune uveitis (EAU) models: a spontaneously relapsing-remitting model and a monophasic/chronic disease model that results in late chorioretinal neovascularization. Both of the diseases are induced by immunization with autoantigen peptides.
METHODS: Prevention was tested using daily oral applications of PP-001 after immunization with the retinal S-antigen peptide PDSAg (for induction of monophasic uveitis and neovascularization) or the interphotoreceptor retinoid-binding protein peptide R14 (for induction of spontaneously relapsing-remitting EAU). Treatment to inhibit relapses and neovascularization was tested using PP-001 daily after the first attack of R14-induced or after onset of PDSAg-induced EAU. Uveitis was graded clinically and histologically. The effect of PP-001 on cytokine secretion and proliferation was evaluated using rat T-cell lines.
RESULTS: Preventive feeding of PP-001 abrogated both types of EAU. Starting treatment after the resolution of the first attack led to a significant reduction of the number and intensity of relapses in R14-induced EAU. PP-001-treatment initiated after onset or after peak of PDSAg-induced EAU significantly reduced neovascularization (as determined by histology). Proliferation of antigen-specific T-cell lines and secretion of IFN-γ, IL-17, IL-10, IP-10, and VEGF were efficiently suppressed by PP-001.
CONCLUSIONS: We investigated a new dihydroorotate dehydrogenase inhibitor as treatment for primary and recurrent disease in relapsing-remitting and chronic rat models of experimental autoimmune uveitis. The small molecule compound PP-001 suppressed proliferation and cytokine secretion of autoreactive T cells (i.e., IFN-g, IL-17, and VEGF) and chorioretinal neovascularization in chronic EAU. Copyright 2015 The Association for Research in Vision and Ophthalmology, Inc.

Entities:  

Keywords:  DHODH inhibitor; chronic EAU; rat model; relapsing-remitting EAU; small molecule

Mesh:

Substances:

Year:  2014        PMID: 25515581     DOI: 10.1167/iovs.14-15518

Source DB:  PubMed          Journal:  Invest Ophthalmol Vis Sci        ISSN: 0146-0404            Impact factor:   4.799


  4 in total

Review 1.  Resolution of uveitis.

Authors:  Gerhild Wildner; Maria Diedrichs-Möhring
Journal:  Semin Immunopathol       Date:  2019-10-07       Impact factor: 9.623

2.  Controlled release of corticosteroid with biodegradable nanoparticles for treating experimental autoimmune uveitis.

Authors:  Lixia Luo; Jin Yang; Yumin Oh; Matthew J Hartsock; Shiyu Xia; Yoo-Chun Kim; Zheng Ding; Tuo Meng; Charles G Eberhart; Laura M Ensign; Jennifer E Thorne; Walter J Stark; Elia J Duh; Qingguo Xu; Justin Hanes
Journal:  J Control Release       Date:  2019-01-17       Impact factor: 9.776

3.  Intraocular DHODH-inhibitor PP-001 suppresses relapsing experimental uveitis and cytokine production of human lymphocytes, but not of RPE cells.

Authors:  Maria Diedrichs-Möhring; Sandy Niesik; Claudia S Priglinger; Stephan R Thurau; Franz Obermayr; Stefan Sperl; Gerhild Wildner
Journal:  J Neuroinflammation       Date:  2018-02-21       Impact factor: 8.322

Review 4.  Multiple sclerosis is linked to MAPKERK overactivity in microglia.

Authors:  George J A Ten Bosch; Jolande Bolk; Bert A 't Hart; Jon D Laman
Journal:  J Mol Med (Berl)       Date:  2021-05-05       Impact factor: 4.599

  4 in total

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