Literature DB >> 25496823

Preliminary evidence of an interaction between the CRISPLD2 gene and non-syndromic cleft lip with or without cleft palate (nsCL/P) in Xinjiang Uyghur population, China.

Ainiwaer Mijiti1, Wang Ling2, Abudukelimujiang Maimaiti3, Maimaitituxun Tuerdi4, Julaiti Tuerxun5, Adili Moming6.   

Abstract

BACKGROUND: Non-syndromic cleft lip with or without cleft palate (nsCL/P) is a common birth defect results from the genetic factors alone or interactions with environmental changes. Single nucleotide polymorphisms (SNPs) of CRISPLD2 gene have been found to be an etiologic factor in the development of nsCL/P. However, few studies to date focused on the association of genetic variation of CRISPLD2 gene with nsCL/P, and the results are conflicting based on the different study population. The main purpose of the present study was to investigate the association between the CRISPLD2 gene and nsCL/P in Xinjiang Uyghur population.
METHODS: Eighteen SNPs were screened in a group of 200 patients with nsCL/P and in a control group consisting of 180 unaffected individuals by next generation sequencing using MiSeq Benchtop Sequencer (Illumina).
RESULTS: Our case-control association analysis showed that the SNP marker rs1546124 showed statistically significant differences in genotype (CC vs. CG vs. GG P=0.004) and allele frequencies (49% vs. 37.8% OR=1.58; 95% CI=1.19-2.1, P=0.002) between nsCL/P and controls. Under the recessive model of inheritance, the GG homozygotes had an OR of 2.4 (95% CI=1.37-4.18; P=0.002), and the result of significance was maintained even after multiple testing correction. Haplotype combinations of CACC were significantly more frequent in the nsCL/P patients than in controls (P=0.037). Finally, the MDR analysis identified the two-SNP model including rs1546124 and rs4782675 as best combination of possibly interactive polymorphisms (P<0.001).
CONCLUSION: Our results demonstrate that genetic polymorphism of CRISPLD2 gene is associated with an increased risk of nsCL/P in a Xinjiang Uyghur population.
Copyright © 2014 Elsevier Ireland Ltd. All rights reserved.

Entities:  

Keywords:  CRISPLD2 gene; Case-control study; Uyghur; nsCL/P

Mesh:

Substances:

Year:  2014        PMID: 25496823     DOI: 10.1016/j.ijporl.2014.10.043

Source DB:  PubMed          Journal:  Int J Pediatr Otorhinolaryngol        ISSN: 0165-5876            Impact factor:   1.675


  4 in total

1.  Crispld2 is required for neural crest cell migration and cell viability during zebrafish craniofacial development.

Authors:  Eric C Swindell; Qiuping Yuan; Lorena E Maili; Bhavna Tandon; Daniel S Wagner; Jacqueline T Hecht
Journal:  Genesis       Date:  2015-09-05       Impact factor: 2.487

2.  Genetic Factors of the Disease Course after Sepsis: A Genome-Wide Study for 28Day Mortality.

Authors:  André Scherag; Franziska Schöneweck; Miriam Kesselmeier; Stefan Taudien; Matthias Platzer; Marius Felder; Christoph Sponholz; Anna Rautanen; Adrian V S Hill; Charles J Hinds; Hamid Hossain; Norbert Suttorp; Oliver Kurzai; Hortense Slevogt; Evangelos J Giamarellos-Bourboulis; Apostolos Armaganidis; Evelyn Trips; Markus Scholz; Frank M Brunkhorst
Journal:  EBioMedicine       Date:  2016-09-15       Impact factor: 8.143

3.  Investigation of candidate genes of non-syndromic cleft lip with or without cleft palate, using both case-control and family-based association studies.

Authors:  Xing Ge; Jia-Wei Hong; Jun-Yu Shen; Zheng Li; Rui Zhang; Qi Wang; Zhen Ding; Gang Chen; Li-Chun Xu
Journal:  Medicine (Baltimore)       Date:  2019-06       Impact factor: 1.817

Review 4.  Assessment of candidate genes and genetic heterogeneity in human non syndromic orofacial clefts specifically non syndromic cleft lip with or without palate.

Authors:  Komal Saleem; Tahir Zaib; Wenjing Sun; Songbin Fu
Journal:  Heliyon       Date:  2019-12-13
  4 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.