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Literature DB >> 25489579

Commentary: CHIPping away pancreatic tumors?

Atul D Joshi¹, John D Catravas¹.

Abstract

Entities: Disease

Year: 2014 PMID： 25489579 PMCID： PMC4245515 DOI： 10.3978/j.issn.2305-5839.2014.11.01

Source DB: PubMed Journal: Ann Transl Med ISSN： 2305-5839

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References

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12 in total

1. Hsp70 and Hsp90 oppositely regulate TGF-β signaling through CHIP/Stub1.

Authors: Yu Shang; Xialian Xu; Xiaolin Duan; Junwei Guo; Yinyin Wang; Fangli Ren; Dacheng He; Zhijie Chang
Journal: Biochem Biophys Res Commun Date: 2014-03-05 Impact factor: 3.575

Review 2. Targeting EGFR in pancreatic cancer treatment.

Authors: T Troiani; E Martinelli; A Capasso; F Morgillo; Michele Orditura; F De Vita; F Ciardiello
Journal: Curr Drug Targets Date: 2012-06 Impact factor: 3.465

3. Balance between folding and degradation for Hsp90-dependent client proteins: a key role for CHIP.

Authors: Lenka Kundrat; Lynne Regan
Journal: Biochemistry Date: 2010-09-07 Impact factor: 3.162

4. Erlotinib prolongs survival in pancreatic cancer by blocking gemcitabine-induced MAPK signals.

Authors: Koji Miyabayashi; Hideaki Ijichi; Dai Mohri; Motohisa Tada; Keisuke Yamamoto; Yoshinari Asaoka; Tsuneo Ikenoue; Keisuke Tateishi; Yousuke Nakai; Hiroyuki Isayama; Yasuyuki Morishita; Masao Omata; Harold L Moses; Kazuhiko Koike
Journal: Cancer Res Date: 2013-02-01 Impact factor: 12.701

Commentary: CHIPping away pancreatic tumors?

1. Hsp70 and Hsp90 oppositely regulate TGF-β signaling through CHIP/Stub1.

Review 2. Targeting EGFR in pancreatic cancer treatment.

3. Balance between folding and degradation for Hsp90-dependent client proteins: a key role for CHIP.

4. Erlotinib prolongs survival in pancreatic cancer by blocking gemcitabine-induced MAPK signals.

Review 5. Therapeutic options for the management of pancreatic cancer.

Review 6. Advances in pancreatic cancer research: moving towards early detection.

Review 7. The ErbB/HER family of protein-tyrosine kinases and cancer.

Review 8. Diverse roles of C-terminal Hsp70-interacting protein (CHIP) in tumorigenesis.

Review 9. The E3 ligase CHIP: insights into its structure and regulation.

10. CHIP is a novel tumor suppressor in pancreatic cancer through targeting EGFR.