Literature DB >> 25440067

Corpora amylacea in the neocortex in patients with temporal lobe epilepsy and focal cortical dysplasia.

B O Estupiñán-Díaz1, L M Morales-Chacón2, I García-Maeso3, L Lorigados-Pedre4, M Báez-Martín2, M E García-Navarro5, O Trápaga-Quincoses2, N Quintanal-Cordero3, J Prince-López3, J E Bender-del Busto6.   

Abstract

INTRODUCTION: Corpora amylacea (CoA) are present in about 60% of atrophic hippocampi resected from patients with drug resistant temporal lobe epilepsy (DRTLE). They have also been described in the lateral temporal neocortex, although less frequently.
OBJECTIVE: The objective is to measure the presence, distribution and density of CoA in the lateral temporal lobes of patients with DRTLE and focal cortical dysplasia (FCD), also examining how CoA density may be linked to demographic and clinical traits.
METHODS: Resected tissue from 35 patients was analysed. CoA density was assessed with a semi-quantitative scale according to the criteria established by Cherian et al.
RESULTS: Presence of CoA in the neocortex of 9 patients was associated with hippocampal sclerosis (FCD type iiia, 7 cases), disembryoplastic neuroepithelial tumour (FCD type iiib, 1 case), and cavernous angioma (FCD type iiic, 1 case). The meningeal surface (MS) was involved in all cases, and 8 cases displayed CoA in the cerebral parenchyma (white matter) and around blood vessels. CoA density on the MS showed a negative correlation with age at seizure onset (r = -0.828, P<.05) and a positive correlation with disease duration (r = 0.678, P<.05) but not with postoperative clinical outcome.
CONCLUSIONS: Patients with DRTLE and a primary lesion (hippocampal sclerosis, tumour, vascular malformation) associated with mild FCD were shown to have CoA deposits in the neocortex. No association was found between presence of CoA and clinical outcome one year after surgery.
Copyright © 2013 Sociedad Española de Neurología. Published by Elsevier Espana. All rights reserved.

Entities:  

Keywords:  Clinical outcome; Corpora amylacea; Cuerpos amiláceos; Displasia cortical focal; Drug-resistant; Epilepsia del lóbulo temporal; Evolución clínica; Farmacorresistencia; Focal cortical dysplasia; Histopathology; Histopatología; Temporal lobe epilepsy

Mesh:

Year:  2014        PMID: 25440067     DOI: 10.1016/j.nrl.2013.06.008

Source DB:  PubMed          Journal:  Neurologia        ISSN: 0213-4853            Impact factor:   3.109


  4 in total

1.  Refractory Epilepsy-MRI, EEG and CT scan, a Correlative Clinical Study.

Authors:  Dijana Nikodijevic; Natalija Baneva-Dolnenec; Dragana Petrovska-Cvetkovska; Daniela Caparoska
Journal:  Open Access Maced J Med Sci       Date:  2016-02-16

2.  Cerebral Corpora amylacea are dense membranous labyrinths containing structurally preserved cell organelles.

Authors:  Paula P Navarro; Christel Genoud; Daniel Castaño-Díez; Alexandra Graff-Meyer; Amanda J Lewis; Yvonne de Gier; Matthias E Lauer; Markus Britschgi; Bernd Bohrmann; Stephan Frank; Jürgen Hench; Gabriel Schweighauser; Annemieke J M Rozemuller; Wilma D J van de Berg; Henning Stahlberg; Sarah H Shahmoradian
Journal:  Sci Rep       Date:  2018-12-21       Impact factor: 4.379

3.  Quantitative analysis of size and regional distribution of corpora amylacea in the hippocampal formation of obstructive sleep apnoea patients.

Authors:  Cuicui Xu; Jessica E Owen; Thorarinn Gislason; Bryndis Benediktsdottir; Stephen R Robinson
Journal:  Sci Rep       Date:  2021-10-22       Impact factor: 4.379

4.  Corpora Amylacea in Neurodegenerative Diseases: Cause or Effect?

Authors:  Troy T Rohn
Journal:  Int J Neurol Neurother       Date:  2015-08-28
  4 in total

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