Literature DB >> 25439493

Identical ATP1A3 mutation causes alternating hemiplegia of childhood and rapid-onset dystonia parkinsonism phenotypes.

Cyrus Boelman1, Ana Marissa Lagman-Bartolome2, Daune L MacGregor2, Jane McCabe2, Willam J Logan2, Berge A Minassian2.   

Abstract

BACKGROUND: Alternating hemiplegia of childhood and rapid-onset dystonia parkinsonism are two separate movement disorders with different dominant mutations in the same sodium-potassium transporter ATPase subunit gene, ATP1A3. PATIENT: We present a child with topiramate-responsive alternating hemiplegia of childhood who was tested for an ATP1A3 gene mutation.
RESULTS: Gene sequencing revealed an identical ATP1A3 mutation as in three typical adult-onset rapid-onset dystonia parkinsonism cases but never previously described in an alternating hemiplegia of childhood case.
CONCLUSION: The discordance of these phenotypes suggests that there are other undiscovered environmental, genetic, or epigenetic factors influencing the development of alternating hemiplegia of childhood or rapid-onset dystonia parkinsonism.
Copyright © 2014 Elsevier Inc. All rights reserved.

Entities:  

Keywords:  ATP1A3; alternating hemiplegia of childhood; genetics; movement disorder; rapid-onset dystonia parkinsonism; topiramate

Mesh:

Substances:

Year:  2014        PMID: 25439493     DOI: 10.1016/j.pediatrneurol.2014.08.015

Source DB:  PubMed          Journal:  Pediatr Neurol        ISSN: 0887-8994            Impact factor:   3.372


  10 in total

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3.  A Portuguese rapid-onset dystonia-parkinsonism case with atypical features.

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  10 in total

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