Literature DB >> 25416713

Blau syndrome: cross-sectional data from a multicentre study of clinical, radiological and functional outcomes.

Carlos D Rosé1, Steven Pans1, Ingele Casteels1, Jordi Anton1, Brigitte Bader-Meunier1, Philippe Brissaud1, Roland Cimaz1, Graciella Espada1, Jorge Fernandez-Martin1, Eric Hachulla1, Miroslav Harjacek1, Raju Khubchandani1, Friederike Mackensen1, Rosa Merino1, Antonio Naranjo1, Sheila Oliveira-Knupp1, Christine Pajot1, Ricardo Russo1, Caroline Thomée1, Sebastiaan Vastert1, Nico Wulffraat1, Juan I Arostegui1, Kevin P Foley1, John Bertin1, Carine H Wouters2.   

Abstract

OBJECTIVE: To report baseline articular, functional and ocular findings of the first international prospective cohort study of Blau syndrome (BS).
METHODS: Three-year, multicentre, observational study on articular, functional (HAQ, Childhood HAQ and VAS global and pain), ophthalmological, therapeutic and radiological data in BS patients.
RESULTS: Baseline data on the first 31 recruited patients (12 females and 19 males) from 18 centres in 11 countries are presented. Of the 31 patients, 11 carried the p.R334W NOD2 mutation, 9 the p.R334Q and 11 various other NOD2 missense mutations; 20 patients were sporadic and 11 from five BS pedigrees. Median disease duration was 12.8 years (1.1-57). Arthritis, documented in all but one patient, was oligoarticular in 7, polyarticular in 23. The median active joint count was 21. Functional capacity was normal in 41%, mildly impaired in 31% and moderate-severe in 28% of patients. The most frequently involved joints at presentation were wrists, ankles, knees and PIPs. On radiographs, a symmetrical non-erosive arthropathy was shown. Previously unknown dysplastic bony changes were found in two-thirds of patients. Ocular disease was documented in 25 of 31 patients, with vitreous inflammation in 64% and moderate-severe visual loss in 33%. Expanded manifestations (visceral, vascular) beyond the classic clinical triad were seen in 52%.
CONCLUSION: BS is associated with severe ocular and articular morbidity. Visceral involvement is common and may be life-threatening. Bone dysplastic changes may show diagnostic value and suggest a previously unknown role of NOD2 in bone morphogenesis. BS is resistant to current drugs, suggesting the need for novel targeted therapies.
© The Author 2014. Published by Oxford University Press on behalf of the British Society for Rheumatology. All rights reserved. For Permissions, please email: journals.permissions@oup.com.

Entities:  

Keywords:  Blau syndrome; NOD2; sarcoidosis; uveitis

Mesh:

Substances:

Year:  2014        PMID: 25416713     DOI: 10.1093/rheumatology/keu437

Source DB:  PubMed          Journal:  Rheumatology (Oxford)        ISSN: 1462-0324            Impact factor:   7.580


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