Literature DB >> 25410145

Elevated uric acid level as a significant predictor of chronic kidney disease: a cohort study with repeated measurements.

Yu-Ching Chou1, Jen-Chun Kuan, Tsan Yang, Wan-Yun Chou, Po-Chien Hsieh, Chyi-Huey Bai, San-Lin You, Chien-Hua Chen, Cheng-Yu Wei, Chien-An Sun.   

Abstract

BACKGROUND: Cohort studies evaluating increased serum uric acid (SUA) level as a chronic kidney disease (CKD) risk factor have yielded variable results. We aimed to assess the association between the pattern of longitudinal changes in SUA and incident CKD [estimated glomerular filtration rate (eGFR) <60 ml/min/1.73 m(2)].
METHODS: A population-based cohort study was conducted on 3,605 participants who were followed prospectively for a mean of 5.18 years. The longitudinal changes in SUA were categorized into three subgroups: persistently low, fluctuated (reduced or elevated), and persistently high. The primary outcome of interest was the development of CKD at a follow-up examination. Cox proportional hazards analysis was used to test the hypothesis.
RESULTS: After adjustment for potential confounders, participants with fluctuated SUA with progressively elevated level and persistently high SUA level had significantly higher risk of developing CKD compared to subjects with persistently low SUA level: adjusted hazard ratio (95% confidence interval) was 2.05 (1.24-3.38) vs. 1.90 (1.34-2.71). This longitudinal relationship was independent of sex, age, body mass index, and hypertension status.
CONCLUSIONS: Longitudinally elevated SUA independently predicts the risk of new-onset CKD.

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Year:  2014        PMID: 25410145     DOI: 10.1007/s40620-014-0158-9

Source DB:  PubMed          Journal:  J Nephrol        ISSN: 1121-8428            Impact factor:   3.902


  23 in total

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5.  Elevated serum uric acid predicts chronic kidney disease.

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7.  Relationship between serum uric acid and clustering of cardiovascular disease risk factors and renal disorders among Shanghai population: a multicentre and cross-sectional study.

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Review 9.  Physiology of Hyperuricemia and Urate-Lowering Treatments.

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10.  Abnormal metabolism of gut microbiota reveals the possible molecular mechanism of nephropathy induced by hyperuricemia.

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