Literature DB >> 25387585

Advanced hepatic fibrosis and steatosis are associated with persistent alanine aminotransferase elevation in chronic hepatitis C patients negative for hepatitis C virus RNA during pegylated interferon plus ribavirin therapy.

Cheng-Chao Liang1, Chen-Hua Liu2, Chen-Shuan Chung3, Cheng-Kuan Lin3, Tung-Hung Su2, Hung-Chih Yang4, Chun-Jen Liu2, Pei-Jer Chen2, Ding-Shinn Chen5, Jia-Horng Kao2.   

Abstract

BACKGROUND: Clinical implications of persistent alanine aminotransferase (ALT) elevation and associated factors in chronic hepatitis C (CHC) patients who achieved undetectable hepatitis C virus (HCV) RNA during pegylated interferon plus ribavirin (peg-IFN/RBV) therapy remain unknown.
METHODS: A total of 1113 CHC patients with undetectable HCV RNA during peg-IFN/RBV therapy were enrolled. Baseline characteristics associated with persistent on-treatment ALT elevation (POAE), and its impact on treatment outcomes, were investigated.
RESULTS: Of 1113 CHC patients, 254 (22.8%) had POAE. Among patients with HCV genotype 1 (HCV-1) who had complete early virologic response (EVR) and received 48 weeks of therapy, patients with POAE had a lower rate of sustained virologic response (SVR) than those without POAE (44.1% vs 74.0%; P = .0002). Multivariate analyses showed that body mass index ≥ 27 kg/m(2), ALT level ≥3 times the upper limit of normal, aspartate aminotransferase to platelet ratio index score ≥1.5, hepatic fibrosis ≥F3, and hepatic steatosis ≥S2 were independent factors associated with POAE after viral clearance.
CONCLUSIONS: POAE is common in CHC patients during therapy. HCV-1 patients with POAE have a lower SVR rate to 48-week therapy if they achieve complete EVR. Advanced hepatic fibrosis, obesity, and steatosis are factors associated with POAE in these patients.
© The Author 2014. Published by Oxford University Press on behalf of the Infectious Diseases Society of America. All rights reserved. For Permissions, please e-mail: journals.permissions@oup.com.

Entities:  

Keywords:  HCV RNA; alanine aminotransferase; chronic hepatitis C; hepatic fibrosis; steatosis

Mesh:

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Year:  2014        PMID: 25387585     DOI: 10.1093/infdis/jiu630

Source DB:  PubMed          Journal:  J Infect Dis        ISSN: 0022-1899            Impact factor:   5.226


  2 in total

Review 1.  PDGF signaling pathway in hepatic fibrosis pathogenesis and therapeutics (Review).

Authors:  Hua-Zhong Ying; Qin Chen; Wen-You Zhang; Huan-Huan Zhang; Yue Ma; Song-Zhao Zhang; Jie Fang; Chen-Huan Yu
Journal:  Mol Med Rep       Date:  2017-09-27       Impact factor: 2.952

2.  Galectin-1 gene silencing inhibits the activation and proliferation but induces the apoptosis of hepatic stellate cells from mice with liver fibrosis.

Authors:  Zhi-Jun Jiang; Qing-Hua Shen; Hai-Yong Chen; Zhe Yang; Ming-Qi Shuai; Shu-Sen Zheng
Journal:  Int J Mol Med       Date:  2018-10-23       Impact factor: 4.101

  2 in total

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