| Literature DB >> 25372919 |
Marcos Aurélio Barboza de Oliveira1, Antônio Carlos Brandi2, Carlos Alberto dos Santos2, Paulo Henrique Husseini Botelho2, José Luis Lasso Cortez3, Domingo Marcolino Braile1.
Abstract
The entry of sodium and calcium play a key effect on myocyte subjected to cardiac arrest by hyperkalemia. They cause cell swelling, acidosis, consumption of adenosine triphosphate and trigger programmed cell death. Cardiac arrest caused by hypocalcemia maintains intracellular adenosine triphosphate levels, improves diastolic performance and reduces oxygen consumption, which can be translated into better protection to myocyte injury induced by cardiac arrest.Entities:
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Year: 2014 PMID： 25372919 PMCID： PMC4412334 DOI： 10.5935/1678-9741.20140074
Source DB: PubMed Journal: Rev Bras Cir Cardiovasc