Literature DB >> 25313913

Prolonged pregnancy in women is associated with attenuated myometrial expression of progesterone receptor co-regulator Krüppel-like Factor 9.

John Mark P Pabona1, Daying Zhang, David S Ginsburg, Frank A Simmen, Rosalia C M Simmen.   

Abstract

CONTEXT: Late-term pregnancy may lead to maternal and neonatal morbidity and mortality. Mice null for the progesterone receptor co-regulator Krüppel-like Factor 9 (KLF9) exhibit delayed parturition and increased incidence of neonatal deaths.
OBJECTIVE: Our aim is to evaluate the contribution of myometrial KLF9 to human parturition.
DESIGN: Myometrial biopsies were obtained from women with term (>37 to ≤41 wk) and late-term (>41 wk) pregnancies during cesarean delivery and assessed for gene and protein expression. Human myometrial cells transfected with nontargeting or KLF9 small interfering RNAs (siRNA) were treated with the progesterone antagonist RU486 and analyzed for pro-inflammatory chemokine/cytokine gene expression.
SETTING: The study took place in a University-affiliated tertiary care hospital and University research laboratory. PATIENTS: Term patients (n = 8) were in spontaneous active labor whereas late-term patients (n = 5) were either in or were induced to active labor, prior to elective cesarean delivery. OUTCOME MEASURES: Steroid hormone receptor, contractility, and inflammation-associated gene expression in myometrial biopsies and in siKLF9-transfected, RU486-treated human myometrial cells was associated with KLF9 expression levels.
RESULTS: Myometrium from women with late-term pregnancy showed lower KLF9, total PGR, and PGR-A/PGR-B isoform expression. Transcript levels of select chemokines/cytokines were up- (CSF3, IL1, IL12A, TGFB2) and down- (CCL3, CCL5, CXCL1, CXCL5, IL15) regulated in late-term relative to term myometrium. Knock-down of KLF9 expression in RU486-treated human myometrial cells modified the expression of PGR and labor-associated cytokines, relative to control siRNA-treated cells.
CONCLUSIONS: Myometrial KLF9 may contribute to the onset of human parturition through its regulation of PGR expression and inflammatory signaling networks.

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Year:  2015        PMID: 25313913      PMCID: PMC4283014          DOI: 10.1210/jc.2014-2846

Source DB:  PubMed          Journal:  J Clin Endocrinol Metab        ISSN: 0021-972X            Impact factor:   5.958


  40 in total

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3.  Progesterone withdrawal, and not increased circulating relaxin, mediates the decrease in myometrial relaxin receptor (RXFP1) expression in late gestation in rats.

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4.  Interleukin-6 is an essential determinant of on-time parturition in the mouse.

Authors:  Sarah A Robertson; Inge Christiaens; Camilla L Dorian; Dean B Zaragoza; Alison S Care; Anke M Banks; David M Olson
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5.  Effects of medroxyprogesterone acetate on gene expression in myometrial explants from pregnant women.

Authors:  Yolande Cordeaux; Mark Tattersall; D Stephen Charnock-Jones; Gordon C S Smith
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6.  Labour is associated with decreased expression of the PGF2alpha receptor (PTGFR) and a novel PTGFR splice variant in human myometrium but not decidua.

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Authors:  John Mark P Pabona; Zhaoyang Zeng; Frank A Simmen; Rosalia C M Simmen
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Review 8.  Minireview: fetal-maternal hormonal signaling in pregnancy and labor.

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9.  p54nrb is a transcriptional corepressor of the progesterone receptor that modulates transcription of the labor-associated gene, connexin 43 (Gja1).

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Authors:  Zhaoyang Zeng; Michael C Velarde; Frank A Simmen; Rosalia C M Simmen
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Review 6.  Transcriptional control of parturition: insights from gene regulation studies in the myometrium.

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7.  Progesterone receptor isoform B regulates the Oxtr-Plcl2-Trpc3 pathway to suppress uterine contractility.

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8.  Evolution of Gene Expression in the Uterine Cervix related to Steroid Signaling: Conserved features in the regulation of cervical ripening.

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Review 9.  Obesity in pregnancy: a novel concept on the roles of adipokines in uterine contractility.

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10.  Extensive shift in placental transcriptome profile in preeclampsia and placental origin of adverse pregnancy outcomes.

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