Literature DB >> 25311781

Does rare matter? Copy number variants at 16p11.2 and the risk of psychosis: a systematic review of literature and meta-analysis.

Giovanni Giaroli1, Nicholas Bass2, Andre Strydom2, Khadijia Rantell2, Andrew McQuillin3.   

Abstract

BACKGROUND: In the last 5 years an increasing number of studies have found that individuals who have micro-duplications at 16p11.2 may have an increased risk of mental disorders including psychotic syndromes.
OBJECTIVE: Our main aim was to review all the evidence in the literature for the association between copy number variants (CNVs) at 16p11.2 and psychosis.
METHODS: We have conducted a systematic review and a meta-analysis utilising the PRISMA statement criteria. We included all original studies (published in English) which presented data on CNVs at 16p11.2 in patients affected by schizophrenia, schizoaffective disorder or bipolar disorder.
RESULTS: We retrieved 15 articles which fulfilled our inclusion criteria. Eleven articles were subsequently selected for a meta-analysis that showed a 10 fold increased risk of psychosis in patients with proximal 16p11.2 duplications. We conducted a second meta-analysis of those studies with low risk of overlap in order to obtain the largest possible sample with the lowest risk of repeated results: 5 studies were selected and we found an odds ratio (OR) of 14.4 (CI=5.2-39.8; p<0.001) for psychosis with proximal 16p11.2 duplications. The results were not significant for micro-deletions in the same region. Finally extracting only those studies that included patients with schizophrenia we found an OR=16.0 (CI=5.4-47.3: p<0.001)
CONCLUSIONS: There is a fourteen fold-increased risk of psychosis and a sixteen fold increased risk of schizophrenia in individuals with micro-duplication at proximal 16p11.2.
Copyright © 2014 The Authors. Published by Elsevier B.V. All rights reserved.

Entities:  

Keywords:  16p11.2; Bipolar disorders; CNV; Copy number variation; Duplication; Psychosis; Schizoaffective disorder; Schizophrenia

Mesh:

Year:  2014        PMID: 25311781     DOI: 10.1016/j.schres.2014.09.025

Source DB:  PubMed          Journal:  Schizophr Res        ISSN: 0920-9964            Impact factor:   4.939


  9 in total

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Authors:  Yingli Fu; Xiaojun Ren; Wei Bai; Qiong Yu; Yaoyao Sun; Yaqin Yu; Na Zhou
Journal:  PeerJ       Date:  2021-08-20       Impact factor: 2.984

2.  16p11.2 deletion is associated with hyperactivation of human iPSC-derived dopaminergic neuron networks and is rescued by RHOA inhibition in vitro.

Authors:  Hannah Pinson; Richard S Smith; Maria Sundberg; Kellen D Winden; Pooja Venugopal; Derek J C Tai; James F Gusella; Michael E Talkowski; Christopher A Walsh; Max Tegmark; Mustafa Sahin
Journal:  Nat Commun       Date:  2021-05-18       Impact factor: 14.919

3.  Linking spatial gene expression patterns to sex-specific brain structural changes on a mouse model of 16p11.2 hemideletion.

Authors:  Vinod Jangir Kumar; Nicola M Grissom; Sarah E McKee; Hannah Schoch; Nicole Bowman; Robbert Havekes; Manoj Kumar; Stephen Pickup; Harish Poptani; Teresa M Reyes; Mike Hawrylycz; Ted Abel; Thomas Nickl-Jockschat
Journal:  Transl Psychiatry       Date:  2018-05-29       Impact factor: 6.222

4.  Refining the Phenotype of Recurrent Rearrangements of Chromosome 16.

Authors:  Serena Redaelli; Silvia Maitz; Francesca Crosti; Elena Sala; Nicoletta Villa; Luigina Spaccini; Angelo Selicorni; Miriam Rigoldi; Donatella Conconi; Leda Dalprà; Gaia Roversi; Angela Bentivegna
Journal:  Int J Mol Sci       Date:  2019-03-04       Impact factor: 5.923

5.  Enzymatic Degradation of Cortical Perineuronal Nets Reverses GABAergic Interneuron Maturation.

Authors:  Ashleigh Willis; Judith A Pratt; Brian J Morris
Journal:  Mol Neurobiol       Date:  2022-03-01       Impact factor: 5.682

6.  Autism Spectrum Disorder, Developmental and Psychiatric Features in 16p11.2 Duplication.

Authors:  LeeAnne Green Snyder; Debra D'Angelo; Qixuan Chen; Raphael Bernier; Robin P Goin-Kochel; Arianne Stevens Wallace; Jennifer Gerdts; Stephen Kanne; Leandra Berry; Lisa Blaskey; Emily Kuschner; Timothy Roberts; Elliot Sherr; Christa L Martin; David H Ledbetter; John E Spiro; Wendy K Chung; Ellen Hanson
Journal:  J Autism Dev Disord       Date:  2016-08

Review 7.  Clinical interpretation of copy number variants in the human genome.

Authors:  Beata Nowakowska
Journal:  J Appl Genet       Date:  2017-09-30       Impact factor: 3.240

Review 8.  Syndromic autism spectrum disorders: moving from a clinically defined to a molecularly defined approach.

Authors:  Bridget A Fernandez; Stephen W Scherer
Journal:  Dialogues Clin Neurosci       Date:  2017-12       Impact factor: 5.986

9.  Genetic copy number variants, cognition and psychosis: a meta-analysis and a family study.

Authors:  Johan H Thygesen; Amelia Presman; Jasmine Harju-Seppänen; Haritz Irizar; Rebecca Jones; Karoline Kuchenbaecker; Kuang Lin; Behrooz Z Alizadeh; Isabelle Austin-Zimmerman; Agna Bartels-Velthuis; Anjali Bhat; Richard Bruggeman; Wiepke Cahn; Stella Calafato; Benedicto Crespo-Facorro; Liewe de Haan; Sonja M C de Zwarte; Marta Di Forti; Álvaro Díez-Revuelta; Jeremy Hall; Mei-Hua Hall; Conrad Iyegbe; Assen Jablensky; Rene Kahn; Luba Kalaydjieva; Eugenia Kravariti; Stephen Lawrie; Jurjen J Luykx; Igancio Mata; Colm McDonald; Andrew M McIntosh; Andrew McQuillin; Rebecca Muir; Roel Ophoff; Marco Picchioni; Diana P Prata; Siri Ranlund; Dan Rujescu; Bart P F Rutten; Katja Schulze; Madiha Shaikh; Frederike Schirmbeck; Claudia J P Simons; Timothea Toulopoulou; Therese van Amelsvoort; Neeltje van Haren; Jim van Os; Ruud van Winkel; Evangelos Vassos; Muriel Walshe; Matthias Weisbrod; Eirini Zartaloudi; Vaughan Bell; John Powell; Cathryn M Lewis; Robin M Murray; Elvira Bramon
Journal:  Mol Psychiatry       Date:  2020-07-27       Impact factor: 15.992

  9 in total

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