Literature DB >> 25301825

Response to letter regarding article, "AT2 receptor activation induces natriuresis and lowers blood pressure".

Brand A Kemp1, Nancy L Howell1, John J Gildea2, Susanna R Keller1, Shetal H Padia1, Robert M Carey1.   

Abstract

Entities:  

Mesh：

Substances：

Year:  2014        PMID： 25301825      PMCID： PMC4296969          DOI： 10.1161/CIRCRESAHA.114.304975

Source DB:  PubMed          Journal:  Circ Res        ISSN： 0009-7330            Impact factor:   17.367


× No keyword cloud information.
  22 in total

1.  Inhibition of pressure natriuresis in mice lacking the AT2 receptor.

Authors:  V Gross; W H Schunck; H Honeck; A F Milia; E Kärgel; T Walther; M Bader; T Inagami; W Schneider; F C Luft
Journal:  Kidney Int       Date:  2000-01       Impact factor: 10.612

2.  Enhancement of angiotensinogen expression in angiotensin II-dependent hypertension.

Authors:  H Kobori; L M Harrison-Bernard; L G Navar
Journal:  Hypertension       Date:  2001-05       Impact factor: 10.190

3.  Selective inhibition of the renal angiotensin type 2 receptor increases blood pressure in conscious rats.

Authors:  A F Moore; N T Heiderstadt; E Huang; N L Howell; Z Q Wang; H M Siragy; R M Carey
Journal:  Hypertension       Date:  2001-05       Impact factor: 10.190

4.  Angiotensin type 2 receptor-mediated hypotension in angiotensin type-1 receptor-blocked rats.

Authors:  R M Carey; N L Howell; X H Jin; H M Siragy
Journal:  Hypertension       Date:  2001-12-01       Impact factor: 10.190

5.  AT₂ receptor activation induces natriuresis and lowers blood pressure.

Authors:  Brandon A Kemp; Nancy L Howell; John J Gildea; Susanna R Keller; Shetal H Padia; Robert M Carey
Journal:  Circ Res       Date:  2014-06-05       Impact factor: 17.367

6.  Sustained hypersensitivity to angiotensin II and its mechanism in mice lacking the subtype-2 (AT2) angiotensin receptor.

Authors:  H M Siragy; T Inagami; T Ichiki; R M Carey
Journal:  Proc Natl Acad Sci U S A       Date:  1999-05-25       Impact factor: 11.205

7.  Protective role of the angiotensin AT2 receptor in a renal wrap hypertension model.

Authors:  H M Siragy; R M Carey
Journal:  Hypertension       Date:  1999-05       Impact factor: 10.190

8.  Angiotensin type 2 receptor mediates valsartan-induced hypotension in conscious rats.

Authors:  H M Siragy; M de Gasparo; R M Carey
Journal:  Hypertension       Date:  2000-05       Impact factor: 10.190

9.  Angiotensin II type 2 receptor overexpression activates the vascular kinin system and causes vasodilation.

Authors:  Y Tsutsumi; H Matsubara; H Masaki; H Kurihara; S Murasawa; S Takai; M Miyazaki; Y Nozawa; R Ozono; K Nakagawa; T Miwa; N Kawada; Y Mori; Y Shibasaki; Y Tanaka; S Fujiyama; Y Koyama; A Fujiyama; H Takahashi; T Iwasaka
Journal:  J Clin Invest       Date:  1999-10       Impact factor: 14.808

10.  AT2 receptor-mediated vasodilatation is unmasked by AT1 receptor blockade in conscious SHR.

Authors:  Xiao C Li; Robert E Widdop
Journal:  Br J Pharmacol       Date:  2004-06-14       Impact factor: 8.739
View more
  3 in total

Review 1.  Genetic and genomic evidence for an important role of the Na+/H+ exchanger 3 in blood pressure regulation and angiotensin II-induced hypertension.

Authors:  Xiao C Li; Xiaowen Zheng; Xu Chen; Chunling Zhao; Dongmin Zhu; Jianfeng Zhang; Jia L Zhuo
Journal:  Physiol Genomics       Date:  2019-03-08       Impact factor: 3.107

Review 2.  The Angiotensin AT2 Receptor: From a Binding Site to a Novel Therapeutic Target.

Authors:  U Muscha Steckelings; Robert E Widdop; Edward D Sturrock; Lizelle Lubbe; Tahir Hussain; Elena Kaschina; Thomas Unger; Anders Hallberg; Robert M Carey; Colin Sumners
Journal:  Pharmacol Rev       Date:  2022-10       Impact factor: 18.923

Review 3.  Brain Angiotensin Type-1 and Type-2 Receptors in Physiological and Hypertensive Conditions: Focus on Neuroinflammation.

Authors:  Khalid Elsaafien; Annette D de Kloet; Eric G Krause; Colin Sumners
Journal:  Curr Hypertens Rep       Date:  2020-07-13       Impact factor: 5.369
  3 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.