Literature DB >> 25265404

Does prenatal diagnosis modify neonatal treatment and early outcome of children with esophageal atresia?

Charles Garabedian1, Rony Sfeir2, Carole Langlois3, Arnaud Bonnard4, Naziha Khen-Dunlop5, Thomas Gelas6, Laurent Michaud2, Fréderic Auber7, Fréderic Gottrand2, Véronique Houfflin-Debarge8.   

Abstract

OBJECTIVE: Our study aimed at (1) evaluating neonatal treatment and outcome of neonates with either a prenatal or a postnatal diagnosis of esophageal atresia (EA) and (2) analyzing the impact of prenatal diagnosis on outcome based on the type of EA. STUDY
DESIGN: We conducted a population-based study using data from the French National Register for infants with EA born from 2008-2010. We compared prenatal, maternal, and neonatal characteristics among children with prenatal vs postnatal diagnosis and EA types I and III. We defined a composite variable of morbidity (anastomotic esophageal leaks, recurrent fistula, stenosis) and death at 1 year.
RESULTS: Four hundred sixty-nine live births with EA were recorded with a prenatal diagnosis rate of 24.3%; 82.2% of EA type I were diagnosed prenatally compared with 17.9% of EA type III (P < .001). Transfer after birth was lower in case of prenatal diagnosis (25.6% vs 82.5%; P < .001). The delay between birth and first intervention did not differ significantly among groups. The defect size was longer among the prenatal diagnosis group (2.61 vs 1.48 cm; P < .001). The composite variables were higher in prenatal diagnosis subset (44% vs 27.6%; P = .003) and in EA type I than in type III (58.1% vs 28.3%; P < .001).
CONCLUSION: Despite the excellent survival rate of EA, cases with antenatal detection have a higher morbidity rate related to the EA type (type I and/or long gap). Even though it does not modify neonatal treatment and the 1-year outcome, prenatal diagnosis allows antenatal parental counselling and avoids postnatal transfers.
Copyright © 2015 Elsevier Inc. All rights reserved.

Entities:  

Keywords:  associated anomaly; esophageal atresia; outcome; postnatal diagnosis; prenatal diagnosis

Mesh:

Year:  2014        PMID: 25265404     DOI: 10.1016/j.ajog.2014.09.030

Source DB:  PubMed          Journal:  Am J Obstet Gynecol        ISSN: 0002-9378            Impact factor:   8.661


  4 in total

1.  Measurement of Gastric Circumference in Foetuses with Oesophageal Atresia.

Authors:  M Hoopmann; K O Kagan; F Borgmeier; G Seitz; J Arand; P Wagner
Journal:  Geburtshilfe Frauenheilkd       Date:  2015-11       Impact factor: 2.915

2.  Type 2 diabetes mellitus induces congenital heart defects in murine embryos by increasing oxidative stress, endoplasmic reticulum stress, and apoptosis.

Authors:  Yanqing Wu; E Albert Reece; Jianxiang Zhong; Daoyin Dong; Wei-Bin Shen; Christopher R Harman; Peixin Yang
Journal:  Am J Obstet Gynecol       Date:  2016-03-31       Impact factor: 8.661

Review 3.  Preoperative management of children with esophageal atresia: current perspectives.

Authors:  Filippo Parolini; Anna Lavinia Bulotta; Sonia Battaglia; Daniele Alberti
Journal:  Pediatric Health Med Ther       Date:  2017-01-18

4.  The upper pouch in oesophageal atresia shows proportional growth during late foetal life.

Authors:  R-B Tröbs; M Nissen; J Wald
Journal:  Acta Paediatr       Date:  2018-05-12       Impact factor: 2.299

  4 in total

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