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Literature DB >> 25224157

Traumatic brain injury: Spreading depolarization can cause secondary injury after TBI.

Alex Chase.

Abstract

Entities: Chemical Disease

Mesh：

Year: 2014 PMID： 25224157 DOI： 10.1038/nrneurol.2014.169

Source DB: PubMed Journal: Nat Rev Neurol ISSN： 1759-4758 Impact factor: 42.937

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2 in total

1. Spreading depression in continuous electroencephalography of brain trauma.

Authors: Jed A Hartings; J Adam Wilson; Jason M Hinzman; Sebastian Pollandt; Jens P Dreier; Vince DiNapoli; David M Ficker; Lori A Shutter; Norberto Andaluz
Journal: Ann Neurol Date: 2014-09-17 Impact factor: 10.422

2. Inverse neurovascular coupling to cortical spreading depolarizations in severe brain trauma.

Authors: Jason M Hinzman; Norberto Andaluz; Lori A Shutter; David O Okonkwo; Clemens Pahl; Anthony J Strong; Jens P Dreier; Jed A Hartings
Journal: Brain Date: 2014-08-24 Impact factor: 13.501

2 in total

1 in total

1. Ca_V2.1 channel mutations causing familial hemiplegic migraine type 1 increase the susceptibility for cortical spreading depolarizations and seizures and worsen outcome after experimental traumatic brain injury.

Authors: Nicole A Terpollili; Reinhard Dolp; Kai Waehner; Susanne M Schwarzmaier; Elisabeth Rumbler; Boyan Todorov; Michel D Ferrari; Arn M J M van den Maagdenberg; Nikolaus Plesnila
Journal: Elife Date: 2022-03-03 Impact factor: 8.140

1 in total

Traumatic brain injury: Spreading depolarization can cause secondary injury after TBI.

1. Spreading depression in continuous electroencephalography of brain trauma.

2. Inverse neurovascular coupling to cortical spreading depolarizations in severe brain trauma.

1. CaV2.1 channel mutations causing familial hemiplegic migraine type 1 increase the susceptibility for cortical spreading depolarizations and seizures and worsen outcome after experimental traumatic brain injury.

1. Ca_V2.1 channel mutations causing familial hemiplegic migraine type 1 increase the susceptibility for cortical spreading depolarizations and seizures and worsen outcome after experimental traumatic brain injury.