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Literature DB >> 25219275

[Mechanisms of hyperphosphorylated tau proteins induce neurotoxicity and traumatic brain injury].

Zi-Ai Zhao, Yuan-Guo Zhou.

Abstract

Entities: Disease

Mesh：

Substances：
tau Proteins

Year: 2014 PMID： 25219275

Source DB: PubMed Journal: Sheng Li Ke Xue Jin Zhan ISSN： 0559-7765

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1 in total

1. Adenosine A_2A receptor inactivation alleviates early-onset cognitive dysfunction after traumatic brain injury involving an inhibition of tau hyperphosphorylation.

Authors: Z-A Zhao; Y Zhao; Y-L Ning; N Yang; Y Peng; P Li; X-Y Chen; D Liu; H Wang; X Chen; W Bai; J-F Chen; Y-G Zhou
Journal: Transl Psychiatry Date: 2017-05-09 Impact factor: 6.222

1 in total

[Mechanisms of hyperphosphorylated tau proteins induce neurotoxicity and traumatic brain injury].

1. Adenosine A2A receptor inactivation alleviates early-onset cognitive dysfunction after traumatic brain injury involving an inhibition of tau hyperphosphorylation.

1. Adenosine A_2A receptor inactivation alleviates early-onset cognitive dysfunction after traumatic brain injury involving an inhibition of tau hyperphosphorylation.