Anne B Skaare1, Anne-Lise Maseng Aas, Nina J Wang. 1. Department of Paediatric Dentistry and Behavioural Science, Institute of Clinical Dentistry, University of Oslo, Oslo, Norway.
Abstract
BACKGROUND: Trauma to primary teeth may have consequences. AIM: To study frequency of enamel defects in permanent successors after luxation injuries, and to report carers' experiences. METHODS: Children 8-15 years (n = 170) suffering luxation injury to primary dentition in 2003 were reexamined in 2010. Permanent successors (n = 300) were clinically examined and photographed. Data from dental records, registration form and a questionnaire were analysed by cross-tabulation and tested by chi-square and t-test. RESULTS: Enamel defects were registered in 130 successor teeth, 22% due to trauma, 21% due to other aetiological factors (MIH, dental fluorosis, idiopathic). Successors with enamel defects were after concussion 8%, subluxation 18%, lateral luxation 41%, intrusion 38% and avulsion 47%. Enamel defects were associated with the child's age and severity of the injury (P < 0.05). Six children had enamel defects in successors of non-injured primary teeth. Anxiety recorded by carers was associated with severity and number of injured teeth (P < 0.05). According to carers eight children developed dental fear, seven were younger than 3.5 years and had had their injured teeth removed. CONCLUSION: Minor luxation injuries and indirect trauma may cause enamel defects in permanent successors. Lower age at injury, severity and number of injured teeth affect carer and child negatively.
BACKGROUND:Trauma to primary teeth may have consequences. AIM: To study frequency of enamel defects in permanent successors after luxation injuries, and to report carers' experiences. METHODS:Children 8-15 years (n = 170) suffering luxation injury to primary dentition in 2003 were reexamined in 2010. Permanent successors (n = 300) were clinically examined and photographed. Data from dental records, registration form and a questionnaire were analysed by cross-tabulation and tested by chi-square and t-test. RESULTS: Enamel defects were registered in 130 successor teeth, 22% due to trauma, 21% due to other aetiological factors (MIH, dental fluorosis, idiopathic). Successors with enamel defects were after concussion 8%, subluxation 18%, lateral luxation 41%, intrusion 38% and avulsion 47%. Enamel defects were associated with the child's age and severity of the injury (P < 0.05). Six children had enamel defects in successors of non-injured primary teeth. Anxiety recorded by carers was associated with severity and number of injured teeth (P < 0.05). According to carers eight children developed dental fear, seven were younger than 3.5 years and had had their injured teeth removed. CONCLUSION: Minor luxation injuries and indirect trauma may cause enamel defects in permanent successors. Lower age at injury, severity and number of injured teeth affect carer and child negatively.