Literature DB >> 25196718

Efficacy of telaprevir-based therapy for difficult-to-treat patients with genotype 2 chronic hepatitis C in Japan.

Hiromitsu Kumada1, Ken Sato2, Tetsuo Takehara3, Makoto Nakamuta4, Masatoshi Ishigami5, Kazuaki Chayama6, Joji Toyota7, Fumitaka Suzuki1, Yoshiyuki Nakayasu8, Miyoko Ochi8, Ichimaro Yamada8, Takeshi Okanoue9.   

Abstract

AIM: This study assessed the efficacy and safety of telaprevir in combination with peginterferon-α-2b (PEG IFN) and ribavirin (RBV), for Japanese difficult-to-treat patients with hepatitis C virus (HCV) genotype 2 who had not achieved sustained virological response (SVR) during prior treatment.
METHODS: In total, 108 relapsed (median age, 59.0 years) and 10 non-responding (median age, 59.0 years) patients with genotype 2 HCV participated. Patients received telaprevir (750 mg, every 8 h) for 12 weeks and PEG IFN/RBV for 24 weeks.
RESULTS: The SVR rates for relapsers and non-responders were 88.0% (95/108) and 50.0% (5/10), respectively. The SVR rates did not differ significantly between patients with rs8099917 TT and non-TT. The SVR rates for relapsers and non-responders with extended rapid viral response (eRVR) were 97.6% (82/84) and 100% (5/5), respectively. On the other hand, the SVR rates for relapsers and non-responders completing the treatment protocol were 98.4% (61/62) and 100% (5/5), respectively. The overall safety profiles of telaprevir-based regimens were similar for Japanese patients with genotype 1 and 2 HCV infection who experienced treatment failure.
CONCLUSION: Telaprevir, in combination with PEG IFN/RBV, provided a high SVR rate for genotype 2 HCV, difficult-to-treat patients who had not achieved SVR during prior IFN-based treatment. The eRVR had a strong influence on the cure rate of telaprevir-based therapy. In addition, the continuation of telaprevir-based treatment for up to 24 weeks was a significant predictor of SVR.
© 2014 The Japan Society of Hepatology.

Entities:  

Keywords:  genotype 2; peginterferon; ribavirin; sustained virological response; telaprevir; treatment failure

Year:  2014        PMID: 25196718     DOI: 10.1111/hepr.12416

Source DB:  PubMed          Journal:  Hepatol Res        ISSN: 1386-6346            Impact factor:   4.288


  4 in total

1.  Substitutions at NS3 Residue 155, 156, or 168 of Hepatitis C Virus Genotypes 2 to 6 Induce Complex Patterns of Protease Inhibitor Resistance.

Authors:  Sanne B Jensen; Stéphanie B N Serre; Daryl G Humes; Santseharay Ramirez; Yi-Ping Li; Jens Bukh; Judith M Gottwein
Journal:  Antimicrob Agents Chemother       Date:  2015-09-21       Impact factor: 5.191

2.  Hepatitis C Virus Genotype 1 to 6 Protease Inhibitor Escape Variants: In Vitro Selection, Fitness, and Resistance Patterns in the Context of the Infectious Viral Life Cycle.

Authors:  Stéphanie B N Serre; Sanne B Jensen; Lubna Ghanem; Daryl G Humes; Santseharay Ramirez; Yi-Ping Li; Henrik Krarup; Jens Bukh; Judith M Gottwein
Journal:  Antimicrob Agents Chemother       Date:  2016-05-23       Impact factor: 5.191

3.  Cost-effectiveness analysis of sofosbuvir plus ribavirin in patients with genotype 2 chronic hepatitis C: an analysis with real world outcomes from a multicentre cohort in Japan.

Authors:  Ataru Igarashi; Norihiro Furusyo; Eiichi Ogawa; Hideyuki Nomura; Kazufumi Dohmen; Nobuhiko Higashi; Kazuhiro Takahashi; Akira Kawano; Koichi Azuma; Takeaki Satoh; Makoto Nakamuta; Toshimasa Koyanagi; Masaki Kato; Shinji Shimoda; Eiji Kajiwara; Jun Hayashi
Journal:  BMJ Open       Date:  2019-06-19       Impact factor: 2.692

4.  Hepatocellular carcinoma or interferon-based therapy history attenuates sofosbuvir/ribavirin for Japanese genotype 2 hepatitis C virus.

Authors:  Masayoshi Yada; Masayuki Miyazaki; Kosuke Tanaka; Akihide Masumoto; Kenta Motomura
Journal:  World J Gastroenterol       Date:  2018-04-07       Impact factor: 5.742

  4 in total

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