Literature DB >> 25171595

Elevated circulating PCSK-9 concentration in renal failure patients is corrected by renal replacement therapy.

Marcin Konarzewski1, Marek Szolkiewicz, Elzbieta Sucajtys-Szulc, Joanna Blaszak, Slawomir Lizakowski, Julian Swierczynski, Boleslaw Rutkowski.   

Abstract

BACKGROUND: A high level of circulating PCSK9 binds to the LDL receptor, reduces its cell's surface density and leads to hypercholesterolemia. The aim of this study was to examine the circulating PCSK9 level in patients with kidney disease.
METHODS: Out of the patients treated in our Departments we selected: (a) 44 patients with CKD stage 3 and 4 (b) 29 patients with CKD stage 5 on maintenance hemodialysis treatment; and (c) 20 patients after successful renal transplantation. Thirty-four subjects, without CKD formed the control group. Serum biochemical parameters' concentrations were assayed by a certified laboratory. Serum PCSK9 concentration was estimated by a commercially available ELISA kit.
RESULTS: The mean serum concentration of PCSK9 in patients with kidney disease was higher than in the control group (238.7 ± 64.5 vs. 536.7 ± 190.4; p < 0.001). A strong negative correlation between serum PCSK9 concentration and eGFR was found (r = -0.66; p < 0.001), as well as between serum concentrations of PCSK9 and total- (r = 0.482; p < 0.05) or LDL-cholesterol (r = 0.533; p < 0.05), but exclusively in patients not receiving statins. The elevated serum concentration of PCSK9 in patients before hemodialysis session declined afterwards, reaching the values observed in patients after kidney transplantation and in the control group.
CONCLUSION: The circulating PCSK9 concentration is increased in patients with CKD; however, this is not accompanied by hypercholesterolemia. The positive correlations between PCSK9/TCh and PCSK9/LDL-Ch have been found only in patients not treated with statins. The elevated circulating PCSK9 level is corrected by maintenance hemodialysis treatment and normalized by a successful kidney transplantation.
© 2014 S. Karger AG, Basel.

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Year:  2014        PMID: 25171595     DOI: 10.1159/000365935

Source DB:  PubMed          Journal:  Am J Nephrol        ISSN: 0250-8095            Impact factor:   3.754


  11 in total

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Journal:  Int Urol Nephrol       Date:  2017-01-13       Impact factor: 2.370

2.  The Role of Proprotein Convertase Subtilisin/Kexin Type 9 in Nephrotic Syndrome-Associated Hypercholesterolemia.

Authors:  Mary E Haas; Amy E Levenson; Xiaowei Sun; Wan-Hui Liao; Joseph M Rutkowski; Sarah D de Ferranti; Valerie A Schumacher; Philipp E Scherer; David J Salant; Sudha B Biddinger
Journal:  Circulation       Date:  2016-07-05       Impact factor: 29.690

Review 3.  Biology of proprotein convertase subtilisin kexin 9: beyond low-density lipoprotein cholesterol lowering.

Authors:  Giuseppe Danilo Norata; Hagai Tavori; Angela Pirillo; Sergio Fazio; Alberico L Catapano
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6.  Hypercholesterolemia in Progressive Renal Failure Is Associated with Changes in Hepatic Heparan Sulfate - PCSK9 Interaction.

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7.  Up-regulation of liver Pcsk9 gene expression as a possible cause of hypercholesterolemia in experimental chronic renal failure.

Authors:  Elzbieta Sucajtys-Szulc; Marek Szolkiewicz; Julian Swierczynski; Boleslaw Rutkowski
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9.  Circulating PCSK9 Level and Risk of Cardiovascular Events and Death in Hemodialysis Patients.

Authors:  Hyeon Seok Hwang; Jin Sug Kim; Yang Gyun Kim; So-Young Lee; Shin Young Ahn; Hong Joo Lee; Dong-Young Lee; Sang Ho Lee; Ju Young Moon; Kyung Hwan Jeong
Journal:  J Clin Med       Date:  2020-01-17       Impact factor: 4.241

10.  Association between PCSK9 Levels and Markers of Inflammation, Oxidative Stress, and Endothelial Dysfunction in a Population of Nondialysis Chronic Kidney Disease Patients.

Authors:  Evangelia Dounousi; Constantinos Tellis; Paraskevi Pavlakou; Anila Duni; Vasillios Liakopoulos; Patrick B Mark; Aikaterini Papagianni; Alexandros D Tselepis
Journal:  Oxid Med Cell Longev       Date:  2021-07-20       Impact factor: 7.310

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