Literature DB >> 25087917

Risk for esophageal neoplasia in Barrett's esophagus patients with mucosal changes indefinite for dysplasia.

Bela Horvath1, Prabhdeep Singh, Hao Xie, Prashanthi N Thota, Daniela S Allende, Rish K Pai, Deepa T Patil, Thomas P Plesec, John R Goldblum, Xiuli Liu.   

Abstract

BACKGROUND AND AIM: Patients with Barrett's esophagus (BE) are at increased risk for esophageal adenocarcinoma (EAC) and therefore require surveillance. Biopsies are classified as indefinite for dysplasia (IND) when the significance of epithelial abnormalities is uncertain due to inflammation or sampling. Our aim was to characterize the neoplastic risk of IND in BE patients and to identify predictors of neoplastic risk.
METHODS: Our pathology database from 1992 to 2007 was searched for BE and IND. Progression rates were calculated and univariate analysis was performed to identify predictors for neoplasia progression in BE-IND patients.
RESULTS: Among 85 patients who had a follow-up (FU) biopsy within 1 year, 11 (12.9%) patients had prevalent neoplasia (seven low-grade dysplasia [LGD], two high-grade dysplasia [HGD], and two EAC). Among 82 patients who did not have prevalent neoplasia but had ≥ 1 year FU, 17 progressed to dysplasia (14 LGD, 3 HGD) and 2 developed EAC during a mean FU period of 59 months. The incidence of neoplasia (LGD, HGD, or EAC) and advanced neoplasia (HGD + EAC) was 4.5 and 1.2 cases per 100 patient-years, respectively. Longer length of BE and multi-focal IND on index biopsy were associated with progression to neoplasia.
CONCLUSION: Patients with BE-IND carry a significant risk of harboring prevalent dysplasia, but the risk of incident dysplasia is similar to the general BE population. The length of BE and the multifocal IND might tentatively help to identify a patient subpopulation at higher risk of neoplastic progression before more definitive data becomes available.
© 2014 Journal of Gastroenterology and Hepatology Foundation and Wiley Publishing Asia Pty Ltd.

Entities:  

Keywords:  Barrett's esophagus; dysplasia; esophageal adenocarcinoma

Mesh:

Year:  2015        PMID: 25087917     DOI: 10.1111/jgh.12696

Source DB:  PubMed          Journal:  J Gastroenterol Hepatol        ISSN: 0815-9319            Impact factor:   4.029


  11 in total

1.  Risk of malignant progression in Barrett's esophagus indefinite for dysplasia.

Authors:  M Ma; S Shroff; M Feldman; M DeMarshall; C Price; A Tierney; G W Falk
Journal:  Dis Esophagus       Date:  2017-03-01       Impact factor: 3.429

Review 2.  Barrett's esophagus in 2016: From pathophysiology to treatment.

Authors:  Irene Martinucci; Nicola de Bortoli; Salvatore Russo; Lorenzo Bertani; Manuele Furnari; Anna Mokrowiecka; Ewa Malecka-Panas; Vincenzo Savarino; Edoardo Savarino; Santino Marchi
Journal:  World J Gastrointest Pharmacol Ther       Date:  2016-05-06

Review 3.  Clinical significance and management of Barrett's esophagus with epithelial changes indefinite for dysplasia.

Authors:  Prashanthi N Thota; Gaurav Kistangari; Ashwini K Esnakula; David Hernandez Gonzalo; Xiu-Li Liu
Journal:  World J Gastrointest Pharmacol Ther       Date:  2016-08-06

Review 4.  Barrett's oesophagus: frequency and prediction of dysplasia and cancer.

Authors:  Gary W Falk
Journal:  Best Pract Res Clin Gastroenterol       Date:  2015-01-20       Impact factor: 3.043

5.  ACG Clinical Guideline: Diagnosis and Management of Barrett's Esophagus.

Authors:  Nicholas J Shaheen; Gary W Falk; Prasad G Iyer; Lauren B Gerson
Journal:  Am J Gastroenterol       Date:  2015-11-03       Impact factor: 10.864

Review 6.  BOB CAT: A Large-Scale Review and Delphi Consensus for Management of Barrett's Esophagus With No Dysplasia, Indefinite for, or Low-Grade Dysplasia.

Authors:  Cathy Bennett; Paul Moayyedi; Douglas A Corley; John DeCaestecker; Yngve Falck-Ytter; Gary Falk; Nimish Vakil; Scott Sanders; Michael Vieth; John Inadomi; David Aldulaimi; Khek-Yu Ho; Robert Odze; Stephen J Meltzer; Eamonn Quigley; Stuart Gittens; Peter Watson; Giovanni Zaninotto; Prasad G Iyer; Leo Alexandre; Yeng Ang; James Callaghan; Rebecca Harrison; Rajvinder Singh; Pradeep Bhandari; Raf Bisschops; Bita Geramizadeh; Philip Kaye; Sheila Krishnadath; M Brian Fennerty; Hendrik Manner; Katie S Nason; Oliver Pech; Vani Konda; Krish Ragunath; Imdadur Rahman; Yvonne Romero; Richard Sampliner; Peter D Siersema; Jan Tack; Tony C K Tham; Nigel Trudgill; David S Weinberg; Jean Wang; Kenneth Wang; Jennie Y Y Wong; Stephen Attwood; Peter Malfertheiner; David MacDonald; Hugh Barr; Mark K Ferguson; Janusz Jankowski
Journal:  Am J Gastroenterol       Date:  2015-04-14       Impact factor: 10.864

7.  Controlled bile acid exposure to oesophageal mucosa causes up-regulation of nuclear γ-H2AX possibly via iNOS induction.

Authors:  Bo Jiang; Shengqian Zhao; Zhen Tao; Jin Wen; Yancheng Yang; Yin Zheng; Hongling Yan; Ying Sheng; Aimin Gao
Journal:  Biosci Rep       Date:  2016-07-07       Impact factor: 3.840

8.  Expression of p53 predicts risk of prevalent and incident advanced neoplasia in patients with Barrett's esophagus and epithelial changes indefinite for dysplasia.

Authors:  Bela Horvath; Prabhdeep Singh; Hao Xie; Prashanthi N Thota; Xingwen Sun; Xiuli Liu
Journal:  Gastroenterol Rep (Oxf)       Date:  2015-10-19

9.  Mutational load may predict risk of progression in patients with Barrett's oesophagus and indefinite for dysplasia: a pilot study.

Authors:  Arvind J Trindade; Matthew J McKinley; Mohammad Alshelleh; Gabriel Levi; Molly Stewart; Kathy J Quinn; Rebecca M Thomas
Journal:  BMJ Open Gastroenterol       Date:  2019-02-02

10.  The risk of neoplasia in patients with Barrett's esophagus indefinite for dysplasia: a multicenter cohort study.

Authors:  Richard Phillips; Wladyslaw Januszewicz; Nastazja D Pilonis; Maria O'Donovan; Tarek Sawas; David A Katzka; Rebecca C Fitzgerald; Massimiliano di Pietro
Journal:  Gastrointest Endosc       Date:  2021-02-04       Impact factor: 9.427

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