Literature DB >> 25019211

TGF β1 mediates epithelial mesenchymal transition via β6 integrin signaling pathway in breast cancer.

Wentong Li1, Baogang Zhang, Hongli Li, Chunling Zhao, Yanmei Zhong, Jing Sun, Shijun Lv.   

Abstract

BACKGROUND: To understand the function of β6 integrin and elucidate its signaling pathways in TGF-β-induced EMT in breast cancer.
METHODS: The interactions between TGF-β1 and β6 integrin were measured by coimmunoprecipitation. The EMT responses, phospherlation of PI3K/Akt and COX-2 expression were determined by real-time PCR, transwell assay, and western blot after the blockage of β6 integrin.
RESULTS: TGF-β1 and β6 integrin could bind with each other. Blockage of β6 integrin rescued TGF-β1-induced EMT phenotype and reduced expression of COX-2 via dephosphorylation of PI3K/Akt.
CONCLUSIONS: β6 integrin plays a critical role in TGF-β1-induced EMT and overexpression of COX-2 in breast cancer.

Entities:  

Keywords:  Breast carcinoma; COX-2; EMT; PI3K pathways; TGF-β; β6 integrin

Mesh:

Substances:

Year:  2014        PMID: 25019211     DOI: 10.3109/07357907.2014.933235

Source DB:  PubMed          Journal:  Cancer Invest        ISSN: 0735-7907            Impact factor:   2.176


  4 in total

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Journal:  Semin Cancer Biol       Date:  2015-05-05       Impact factor: 15.707

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Journal:  Int J Mol Sci       Date:  2018-03-29       Impact factor: 5.923

3.  LncRNA-AC009948.5 promotes invasion and metastasis of lung adenocarcinoma by binding to miR-186-5p.

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Review 4.  The Inflammatory Profile of the Tumor Microenvironment, Orchestrated by Cyclooxygenase-2, Promotes Epithelial-Mesenchymal Transition.

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  4 in total

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