Literature DB >> 24989722

Smoking behaviour modifies IL23r-associated disease risk in patients with Crohn's disease.

James D Doecke1, Lisa A Simms, Zhen Zhen Zhao, Rebecca L Roberts, Elizabeth V Fowler, Anthony Croft, Angela Lin, Ning Huang, David C Whiteman, Timothy H J Florin, Murray L Barclay, Tony R Merriman, Richard B Gearry, Grant W Montgomery, Graham L Radford-Smith.   

Abstract

BACKGROUND AND AIM: The etiology of Crohn's disease (CD) implicates both genetic and environmental factors. Smoking behavior is one environmental risk factor to play a role in the development of CD. The study aimed to assess the contribution of the interleukin 23 receptor (IL23R) in determining disease susceptibility in two independent cohorts of CD, and to investigate the interactions between IL23R variants, smoking behavior, and CD-associated genes, NOD2 and ATG16L1.
METHODS: Ten IL23R single-nucleotide polymorphisms (SNPs) were genotyped in 675 CD cases, and 1255 controls from Brisbane, Australia (dataset 1). Six of these SNPs were genotyped in 318 CD cases and 533 controls from Canterbury, New Zealand (dataset 2). Case-control analysis of genotype and allele frequencies, and haplotype analysis for all SNPs was conducted.
RESULTS: We demonstrate a strong increased CD risk for smokers in both datasets (odds ratio 3.77, 95% confidence interval 2.88-4.94), and an additive interaction between IL23R SNPs and cigarette smoking. Ileal involvement was a consistent marker of strong SNP-CD association (P ≤ 0.001), while the lowest minor allele frequencies for location were found in those with colonic CD (L2). Three haplotype blocks were identified across the 10 IL23R SNPs conferring different risk of CD. Haplotypes conferred no further risk of CD when compared with single SNP analyses.
CONCLUSION: IL23R gene variants determine CD susceptibility in the Australian and New Zealand population, particularly ileal CD. A strong additive interaction exists between IL23R SNPs and smoking behavior resulting in a dramatic increase in disease risk depending upon specific genetic background.
© 2014 Journal of Gastroenterology and Hepatology Foundation and Wiley Publishing Asia Pty Ltd.

Entities:  

Keywords:  ATG16L1; Crohn's disease; IL23R; NOD2; interleukin 23 receptor; smoking

Mesh:

Substances:

Year:  2015        PMID: 24989722     DOI: 10.1111/jgh.12674

Source DB:  PubMed          Journal:  J Gastroenterol Hepatol        ISSN: 0815-9319            Impact factor:   4.029


  8 in total

1.  Genetic Factors Interact With Tobacco Smoke to Modify Risk for Inflammatory Bowel Disease in Humans and Mice.

Authors:  Pankaj Yadav; David Ellinghaus; Gaëlle Rémy; Sandra Freitag-Wolf; Anabelle Cesaro; Frauke Degenhardt; Gabrielle Boucher; Myriam Delacre; Laurent Peyrin-Biroulet; Muriel Pichavant; John D Rioux; Philippe Gosset; Andre Franke; L Philip Schumm; Michael Krawczak; Mathias Chamaillard; Astrid Dempfle; Vibeke Andersen
Journal:  Gastroenterology       Date:  2017-05-12       Impact factor: 22.682

Review 2.  ACG Clinical Guideline: Preventive Care in Inflammatory Bowel Disease.

Authors:  Francis A Farraye; Gil Y Melmed; Gary R Lichtenstein; Sunanda V Kane
Journal:  Am J Gastroenterol       Date:  2017-01-10       Impact factor: 10.864

3.  Role of environmental factors in the pathogenesis of Crohn's disease: a critical review.

Authors:  Yueying Chen; Yining Wang; Jun Shen
Journal:  Int J Colorectal Dis       Date:  2019-11-16       Impact factor: 2.571

Review 4.  The NOD2-Smoking Interaction in Crohn's Disease is likely Specific to the 1007fs Mutation and may be Explained by Age at Diagnosis: A Meta-Analysis and Case-Only Study.

Authors:  M Ellen Kuenzig; Jeff Yim; Stephanie Coward; Bertus Eksteen; Cynthia H Seow; Cheryl Barnabe; Herman W Barkema; Mark S Silverberg; Peter L Lakatos; Paul L Beck; Richard Fedorak; Levinus A Dieleman; Karen Madsen; Remo Panaccione; Subrata Ghosh; Gilaad G Kaplan
Journal:  EBioMedicine       Date:  2017-06-16       Impact factor: 8.143

5.  Smoking Interacts With CHRNA5, a Nicotinic Acetylcholine Receptor Subunit Gene, to Influence the Risk of IBD-Related Surgery.

Authors:  Kelly C Cushing; Adeeti Chiplunker; Allie Li; Yun Ju Sung; Taylor Geisman; Li-Shiun Chen; Sharon Cresci; Alexandra M Gutierrez
Journal:  Inflamm Bowel Dis       Date:  2018-04-23       Impact factor: 5.325

6.  A Gene-Environment Interaction Between Smoking and Gene polymorphisms Provides a High Risk of Two Subgroups of Sarcoidosis.

Authors:  Natalia V Rivera; Karina Patasova; Susanna Kullberg; Lina Marcela Diaz-Gallo; Tomoko Iseda; Camilla Bengtsson; Lars Alfredsson; Anders Eklund; Ingrid Kockum; Johan Grunewald; Leonid Padyukov
Journal:  Sci Rep       Date:  2019-12-09       Impact factor: 4.379

7.  Association of Interleukin-23 receptor gene polymorphisms with susceptibility to Crohn's disease: A meta-analysis.

Authors:  Wang-Dong Xu; Qi-Bing Xie; Yi Zhao; Yi Liu
Journal:  Sci Rep       Date:  2015-12-18       Impact factor: 4.379

8.  Relationship between the IL23R SNPs and Crohn's Disease Susceptibility and Phenotype in the Polish and Bosnian Populations: A Case-Control Study.

Authors:  Krzysztof Borecki; Iwona Zawada; Nermin Nusret Salkić; Beata Karakiewicz; Grażyna Adler
Journal:  Int J Environ Res Public Health       Date:  2019-05-02       Impact factor: 3.390

  8 in total

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