Literature DB >> 2496362

Proto-oncogene c-fos is transiently induced in the rat cerebral cortex after forebrain ischemia.

H Onodera1, K Kogure, Y Ono, K Igarashi, Y Kiyota, A Nagaoka.   

Abstract

The amounts of mRNAs for proto-oncogene c-fos and structural protein beta-actin were measured in the rat cerebral cortex after transient forebrain ischemia. A transient and specific induction of c-fos mRNA was noticed in the cerebral cortex 30-90 min after ischemia followed by decline to control value. In contrast, the level of mRNA for beta-actin was not altered throughout the recirculation period examined. These results suggest specific role of c-fos gene after brain damage.

Entities:  

Mesh：

Substances：

Year:  1989        PMID： 2496362     DOI： 10.1016/0304-3940(89)90381-9

Source DB:  PubMed          Journal:  Neurosci Lett        ISSN： 0304-3940            Impact factor:   3.046


  11 in total

Review 1.  Immediate early gene expression within the visual system: light and circadian regulation in the retina and the suprachiasmatic nucleus.

Authors:  B L Caputto; M E Guido
Journal:  Neurochem Res       Date:  2000-01       Impact factor: 3.996

2.  Opposite effects of rough and gentle handling with repeated saline administration on c-fos mRNA expression in the rat brain.

Authors:  M Asanuma; N Ogawa; H Hirata; H Chou; K Tanaka; A Mori
Journal:  J Neural Transm Gen Sect       Date:  1992

Review 3.  Excitable membranes, lipid messengers, and immediate-early genes. Alteration of signal transduction in neuromodulation and neurotrauma.

Authors:  J P Doucet; N G Bazan
Journal:  Mol Neurobiol       Date:  1992       Impact factor: 5.590

Review 4.  Fos-jun and the primary genomic response in the nervous system. Possible physiological role and pathophysiological significance.

Authors:  J P Doucet; S P Squinto; N G Bazan
Journal:  Mol Neurobiol       Date:  1990 Spring-Summer       Impact factor: 5.590

5.  Brainstem ischemic damage following occlusion of the blood vessels in the rat's posterior cerebral circulation.

Authors:  H Inui; T Murai; K Yane; T Matsunaga
Journal:  Eur Arch Otorhinolaryngol       Date:  1996       Impact factor: 2.503

6.  MK-801 prevents cyanide-induced changes of Fos levels in rat brain.

Authors:  G Pavlaković; A Rathinavelu; G E Isom
Journal:  Neurochem Res       Date:  1994-10       Impact factor: 3.996

7.  Muscarinic cholinergic receptor-mediated modulation on striatal c-fos mRNA expression induced by levodopa in rat brain.

Authors:  H Chou; N Ogawa; M Asanuma; H Hirata; A Mori
Journal:  J Neural Transm Gen Sect       Date:  1992

8.  Neocortical spreading depression provokes the expression of c-fos protein-like immunoreactivity within trigeminal nucleus caudalis via trigeminovascular mechanisms.

Authors:  M A Moskowitz; K Nozaki; R P Kraig
Journal:  J Neurosci       Date:  1993-03       Impact factor: 6.167

9.  Ubiquitin proteasome-mediated synaptic reorganization: a novel mechanism underlying rapid ischemic tolerance.

Authors:  Robert Meller; Simon John Thompson; Theresa Ann Lusardi; Andrea Nicole Ordonez; Michelle Dawn Ashley; Veronica Jessick; Weihzen Wang; Daniel John Torrey; David Clifford Henshall; Philip R Gafken; Julie Anne Saugstad; Zhi-Gang Xiong; Roger Pancoast Simon
Journal:  J Neurosci       Date:  2008-01-02       Impact factor: 6.167

10.  Effects of Ca2+ channel blockers on cortical hypoperfusion and expression of c-Fos-like immunoreactivity after cortical spreading depression in rats.

Authors:  M Shimazawa; H Hara; T Watano; T Sukamoto
Journal:  Br J Pharmacol       Date:  1995-08       Impact factor: 8.739
View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.