Literature DB >> 24962191

Enteral nutrition and total parenteral nutrition components in the course of total parenteral nutrition-associated cholestasis in neonatal necrotizing enterocolitis.

Michelle Veenstra1, Logan Danielson2, Evan Brownie2, May Saba3, Girija Natarajan4, Michael Klein5.   

Abstract

BACKGROUND: Newborns with necrotizing enterocolitis (NEC) are at high risk for the development of total parenteral nutritional-associated cholestasis (TPNAC). Patients with NEC were evaluated to determine risk factors for development of TPNAC and predictors of resolution. We hypothesized that there are additional factors relating to the timing of enteral nutrition or TPN components that effect development and persistence of TPNAC in patients with NEC that may be altered to decrease the chance of progression to liver failure.
METHODS: This was a retrospective chart review of NEC patients from 2001 to 2010. TPNAC was defined as direct bilirubin ≥2 mg/dL, the level used for cholestasis in neonatal studies relating to TPNAC.
RESULTS: Of 178 patients with NEC, 96 developed TPNAC, and in 27 TPNAC had resolved by discharge. Days of TPN did not affect TPNAC resolution by discharge (P = 1.0). TPNAC was less likely to occur in patients with body weights >1,500 g, enteral nutrition within the first week of life, no infection, fewer TPN days, and lesser hospital stay (P < .01). There were many factors affecting resolution of cholestasis. Enteral nutrition within 6 days of birth decreased development of TPNAC (P < .01), and resumption of enteral nutrition within 3 weeks after NEC diagnosis increased the resolution of cholestasis (P < .01). No component of TPN was important for the development or resolution of cholestasis.
CONCLUSION: Of the factors that effect development and resolution of TPNAC in NEC, the ones that we can alter include early enteral feeds and surveillance for infection.
Copyright © 2014 Mosby, Inc. All rights reserved.

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Year:  2014        PMID: 24962191     DOI: 10.1016/j.surg.2014.04.031

Source DB:  PubMed          Journal:  Surgery        ISSN: 0039-6060            Impact factor:   3.982


  4 in total

1.  The miR-223/nuclear factor I-A axis regulates inflammation and cellular functions in intestinal tissues with necrotizing enterocolitis.

Authors:  Yu Zheng Wu; Kathy Yuen Yee Chan; Kam Tong Leung; Hugh Simon Lam; Yuk Him Tam; Kim Hung Lee; Karen Li; Pak Cheung Ng
Journal:  FEBS Open Bio       Date:  2021-06-01       Impact factor: 2.693

2.  Parenteral Nutrition-Associated Cholestasis in Very Low Birth Weight Infants: A Single Center Experience.

Authors:  Ah-Young Kim; Ryoung-Kyoung Lim; Young-Mi Han; Kyung-Hee Park; Shin-Yun Byun
Journal:  Pediatr Gastroenterol Hepatol Nutr       Date:  2016-03-22

3.  Aggressive nutrition in extremely low birth weight infants: impact on parenteral nutrition associated cholestasis and growth.

Authors:  Andreas Repa; Ruth Lochmann; Lukas Unterasinger; Michael Weber; Angelika Berger; Nadja Haiden
Journal:  PeerJ       Date:  2016-09-20       Impact factor: 2.984

4.  Immediate Post-operative Enterocyte Injury, as Determined by Increased Circulating Intestinal Fatty Acid Binding Protein, Is Associated With Subsequent Development of Necrotizing Enterocolitis After Infant Cardiothoracic Surgery.

Authors:  John D Watson; Tracy T Urban; Suhong S Tong; Jeanne Zenge; Ludmilla Khailova; Paul E Wischmeyer; Jesse A Davidson
Journal:  Front Pediatr       Date:  2020-05-27       Impact factor: 3.418

  4 in total

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