Literature DB >> 24950695

Silencing of CCR2 in myocarditis.

Florian Leuschner1, Gabriel Courties2, Partha Dutta2, Luke J Mortensen2, Rostic Gorbatov2, Brena Sena2, Tatiana I Novobrantseva3, Anna Borodovsky3, Kevin Fitzgerald3, Victor Koteliansky4, Yoshiko Iwamoto2, Marina Bohlender5, Soeren Meyer5, Felix Lasitschka6, Benjamin Meder5, Hugo A Katus5, Charles Lin2, Peter Libby7, Filip K Swirski2, Daniel G Anderson8, Ralph Weissleder9, Matthias Nahrendorf10.   

Abstract

BACKGROUND: Myocarditis is characterized by inflammatory cell infiltration of the heart and subsequent deterioration of cardiac function. Monocytes are the most prominent population of accumulating leucocytes. We investigated whether in vivo administration of nanoparticle-encapsulated siRNA targeting chemokine (C-C motif) receptor 2 (CCR2)-a chemokine receptor crucial for leucocyte migration in humans and mice--reduces inflammation in autoimmune myocarditis. METHODS AND
RESULTS: In myocardium of patients with myocarditis, CCL2 mRNA levels and CCR2(+) cells increased (P < 0.05), motivating us to pursue CCR2 silencing. Flow cytometric analysis showed that siRNA silencing of CCR2 (siCCR2) reduced the number of Ly6C(high) monocytes in hearts of mice with acute autoimmune myocarditis by 69% (P < 0.05), corroborated by histological assessment. The nanoparticle-delivered siRNA was not only active in monocytes but also in bone marrow haematopoietic progenitor cells. Treatment with siCCR2 reduced the migration of bone marrow granulocyte macrophage progenitors into the blood. Cellular magnetic resonance imaging (MRI) after injection of macrophage-avid magnetic nanoparticles detected myocarditis and therapeutic effects of RNAi non-invasively. Mice with acute myocarditis showed enhanced macrophage MRI contrast, which was prevented by siCCR2 (P < 0.05). Follow-up MRI volumetry revealed that siCCR2 treatment improved ejection fraction (P < 0.05 vs. control siRNA-treated mice).
CONCLUSION: This study highlights the importance of CCR2 in the pathogenesis of myocarditis. In addition, we show that siCCR2 affects leucocyte progenitor trafficking. The data also point to a novel therapeutic strategy for the treatment of myocarditis. Published on behalf of the European Society of Cardiology. All rights reserved.
© The Author 2014. For permissions please email: journals.permissions@oup.com.

Entities:  

Keywords:  Inflammation; Molecular imaging; Myocarditis; Nanoparticle; siRNA

Mesh:

Substances:

Year:  2014        PMID: 24950695      PMCID: PMC4465633          DOI: 10.1093/eurheartj/ehu225

Source DB:  PubMed          Journal:  Eur Heart J        ISSN: 0195-668X            Impact factor:   29.983


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