Literature DB >> 24840536

Transient increase in circulating myeloid-derived suppressor cells after partial bladder outlet obstruction.

Wei-Yu Lin1, Ching Chuan Hsieh1, Teng-Yao Yang1, Miaw-Ling Chen1, Li Ying Huang1, Yi-Pai Lin1, Pey-Jium Chang1, Robert M Levin1, Yau-Huei Wei1.   

Abstract

PURPOSE: Partial bladder outlet obstruction causes a significant increase in tissue and systemic oxidative stress markers and tissue inflammatory cytokine levels. Myeloid-derived suppressor cells, IFN-γ, IL-10 and aldosterone are believed to be associated with oxidative stress and inflammation. We investigated alterations in plasma myeloid-derived suppressor cells, IFN-γ, IL-10 and aldosterone levels in partial bladder outlet obstruction and after its reversal.
MATERIALS AND METHODS: Rats with surgically induced partial bladder outlet obstruction were divided into 4 groups of 3 each, including sham treated, 4-week obstruction, and 4 and 8-week obstruction with relief. Plasma levels of circulating myeloid-derived suppressor cells, IFN-γ, IL-10 and aldosterone were assessed by flow cytometry or enzyme-linked immunosorbent assay.
RESULTS: The circulating myeloid-derived suppressor cell level was markedly increased in the obstruction group compared to the sham treated group and it returned to normal in the 4 and 8-week obstruction with relief groups. Plasma IFN-γ, IL-10 and aldosterone were similarly increased in the obstruction group and returned to normal in the 4 and 8-week obstruction with relief groups.
CONCLUSIONS: Levels of circulating myeloid-derived suppressor cells, IFN-γ, IL-10 and aldosterone were increased in rats with partial bladder outlet obstruction but returned to normal after reversal. This suggests that an increase in these parameters may be a good predictive indicator of patients at increased risk for urinary symptoms.
Copyright © 2014 American Urological Association Education and Research, Inc. Published by Elsevier Inc. All rights reserved.

Entities:  

Keywords:  cell differentiation; cytokines; inflammation; oxidative stress; urinary bladder neck obstruction

Mesh:

Substances:

Year:  2014        PMID: 24840536     DOI: 10.1016/j.juro.2014.05.045

Source DB:  PubMed          Journal:  J Urol        ISSN: 0022-5347            Impact factor:   7.450


  3 in total

1.  New frontiers in molecular and imaging research on evaluation and diagnosis of bladder outlet obstruction in women.

Authors:  Laura Martinez; Rose Khavari
Journal:  Curr Bladder Dysfunct Rep       Date:  2017-08-04

Review 2.  Bladder Hyperactivity Induced by Oxidative Stress and Bladder Ischemia: A Review of Treatment Strategies with Antioxidants.

Authors:  Yi-Hsuan Wu; Kuang-Shun Chueh; Shu-Mien Chuang; Cheng-Yu Long; Jian-He Lu; Yung-Shun Juan
Journal:  Int J Mol Sci       Date:  2021-06-02       Impact factor: 5.923

3.  Human amniotic fluid stem cells can alleviate detrusor dysfunction caused by bladder outlet obstruction in rats.

Authors:  Ching-Chung Liang; Wen-Chu Huang; Steven W Shaw; Yung-Hsin Huang; Tsong-Hai Lee
Journal:  Sci Rep       Date:  2022-04-23       Impact factor: 4.996

  3 in total

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