Literature DB >> 24815134

Ischemic reperfusion injury-induced oxidative stress and pro-inflammatory mediators in liver transplantation recipients.

Y-F Tsai1, F-C Liu1, W-C Sung2, C-C Lin1, P C-H Chung3, W-C Lee4, H-P Yu5.   

Abstract

OBJECTIVE: Liver ischemic reperfusion injury is harmful to transplant recipients, and is associated with postoperative morbidity and mortality. Our study was designed to investigate the oxidative stress and pro-inflammatory mediators in liver transplant recipients.
METHODS: We prospectively analyzed 14 recipients who underwent liver transplantation by measuring their blood levels of malondialdehyde (MDA) and cytokines, such as tumor necrosis factor-α (TNF-α), interleukin-1β (IL-1β), and IL-6, at nine time points perioperatively. We also evaluated the correlations between oxidative stress (MDA levels) and the characteristics of the recipient or the donated graft.
RESULTS: These parameters significantly increased from 1 minute before reperfusion, and the values peaked within 3 to 30 minutes after reperfusion. On the time point at 5 minutes after reperfusion, the MDA levels which were the highest in the recipients correlated with the values of preoperative direct/and total bilirubin, aspartate aminotransferase (AST), alanine aminotransferase (ALT), model for end-stage liver disease (MELD) score, international normalized ratio (INR), and surgical blood loss.
CONCLUSION: The levels of MDA, TNF-α, IL-1β, and IL-6 greatly increased with the ischemic reperfusion insult. Recipients with higher values of preoperative direct/and total bilirubin, AST, ALT, MELD score, INR, and surgical blood loss tended to have higher levels of MDA and may suffer more injury from this insult.
Copyright © 2014 Elsevier Inc. All rights reserved.

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Year:  2014        PMID: 24815134     DOI: 10.1016/j.transproceed.2014.01.009

Source DB:  PubMed          Journal:  Transplant Proc        ISSN: 0041-1345            Impact factor:   1.066


  5 in total

Review 1.  Post reperfusion syndrome during liver transplantation: From pathophysiology to therapy and preventive strategies.

Authors:  Antonio Siniscalchi; Lorenzo Gamberini; Cristiana Laici; Tommaso Bardi; Giorgio Ercolani; Laura Lorenzini; Stefano Faenza
Journal:  World J Gastroenterol       Date:  2016-01-28       Impact factor: 5.742

2.  Ursolic acid inhibits superoxide production in activated neutrophils and attenuates trauma-hemorrhage shock-induced organ injury in rats.

Authors:  Tsong-Long Hwang; Hsin-I Shen; Fu-Chao Liu; Hsin-I Tsai; Yang-Chang Wu; Fang-Rong Chang; Huang-Ping Yu
Journal:  PLoS One       Date:  2014-10-31       Impact factor: 3.240

3.  Farnesoid X Receptor Activation Protects Liver From Ischemia/Reperfusion Injury by Up-Regulating Small Heterodimer Partner in Kupffer Cells.

Authors:  Dan Jin; Tianfei Lu; Ming Ni; Han Wang; Jiang Zhang; Chenpeng Zhong; Chuan Shen; Jun Hao; Ronald W Busuttil; Jerzy W Kupiec-Weglinski; Jianjun Zhang; Ning Xu; Yuan Zhai
Journal:  Hepatol Commun       Date:  2020-02-13

4.  5-Aminolevulinate improves metabolic recovery and cell survival of the liver following cold preservation.

Authors:  Xiaomei Zhang; Liang Chen; Wei Liu; Juan Shen; Haobin Sun; Jinliang Liang; Guo Lv; Guihua Chen; Yang Yang; Jingxing Ou
Journal:  Theranostics       Date:  2022-03-21       Impact factor: 11.600

Review 5.  ROS homeostasis, a key determinant in liver ischemic-preconditioning.

Authors:  Ignacio Prieto; María Monsalve
Journal:  Redox Biol       Date:  2017-05-04       Impact factor: 11.799

  5 in total

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