Literature DB >> 24801669

Targeting angiogenesis as a therapeutic means to reinforce osteocyte survival and prevent nonunions in the aftermath of radiotherapy.

Alexis Donneys1, Noah S Nelson1, Erin E Page1, Sagar S Deshpande1, Peter A Felice1,2, Catherine N Tchanque-Fossuo1, Joshua P Spiegel1, Steven R Buchman1.   

Abstract

BACKGROUND: Radiotherapy (XRT) exerts detrimental collateral effects on bone tissue through mechanisms of vascular damage and impediments to osteocytes, ultimately predisposing patients to the debilitating problems of late pathologic fractures and nonunions. We posit that angiogenic therapy will reverse these pathologic effects in a rat model of radiated fracture healing.
METHODS: Three groups of rats underwent mandibular osteotomy. Radiated groups received a fractionated 35-Gy dose before surgery. The deferoxamine (DFO) group received local injections postoperatively. A 40-day healing period was allowed before histology. Analysis of variance (ANOVA; p < .05) was used for group comparisons.
RESULTS: Radiated fractures revealed a significantly decreased osteocyte count and corresponding increase in empty lacunae when compared to nonradiated fractures (p = .001). With the addition of DFO, these differences were not appreciated. Further, a 42% increase in bony unions was observed after DFO therapy.
CONCLUSION: Targeting angiogenesis is a useful means for promoting osteocyte survival and preventing bone pathology after XRT.
© 2014 Wiley Periodicals, Inc.

Entities:  

Keywords:  angiogenesis; deferoxamine; mandible; nonunion; radiation

Mesh:

Substances:

Year:  2014        PMID: 24801669      PMCID: PMC4788100          DOI: 10.1002/hed.23744

Source DB:  PubMed          Journal:  Head Neck        ISSN: 1043-3074            Impact factor:   3.147


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