Literature DB >> 24797963

Increased human leukocyte antigen-G expression at the maternal-fetal interface is associated with preterm birth.

Molly J Stout1, Bin Cao, Michele Landeau, Jacob French, George A Macones, Indira U Mysorekar.   

Abstract

UNLABELLED: Abstract Objective: The maternal-fetal interface must modulate immune function to allow tolerance of fetal cells while still reacting to pathogens to suppress infection. Human leukocyte antigen-G (HLA-G) is a class Ib major histocompatibility complex protein involved in maternal-fetal tolerance. We posited that alterations in placental HLA-G expression predispose women to preterm birth. The aim of this study was to compare HLA-G expression in the maternal-fetal interface of term versus preterm human placentas.
METHODS: We performed a cross-sectional study of specimens from the basal plate of the human placenta from women enrolled in a tissue specimen and clinical data consortium. Immunohistochemistry with digital microscopic analysis was used to quantify HLA-G protein expression in the basal plate from preterm and term placentas.
RESULTS: Preterm birth <37 weeks occurred in 29.5% of 149 singleton pregnancies. HLA-G-positive cells occupied one-third of the basal plates, and the HLA-G-positive area was increased by 14% in placentas from preterm births than in those from term births (32.1% in term placentas versus 36.6% in preterm placentas).
CONCLUSION: Although HLA-G is required for maternal tolerance of the semi-allogeneic fetus, higher levels of HLA-G expression at the maternal-fetal interface is associated with preterm birth.

Entities:  

Keywords:  Extravillous trophoblast; infection; maternal–fetal tolerance; placenta

Mesh:

Substances:

Year:  2014        PMID: 24797963      PMCID: PMC4247801          DOI: 10.3109/14767058.2014.921152

Source DB:  PubMed          Journal:  J Matern Fetal Neonatal Med        ISSN: 1476-4954


  38 in total

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