Literature DB >> 2478818

Regulation of smooth muscle cell growth in injured artery.

A W Clowes1, M M Clowes, J Fingerle, M A Reidy.   

Abstract

The process of intimal thickening after arterial injury can be divided into several steps, namely, initiation of smooth muscle cell proliferation, migration, and further intimal proliferation and deposition of matrix. Factors controlling the initiation of proliferation include de-endothelialization and vessel distension but not platelet adherence. Migration and subsequent intimal proliferation are controlled by factors from platelets, re-endothelialization, and endogenously released gamma-interferon.

Mesh：

Year:  1989        PMID： 2478818

Source DB:  PubMed          Journal:  J Cardiovasc Pharmacol        ISSN： 0160-2446            Impact factor:   3.105


  29 in total

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6.  Periadventitial adipose tissue modulates the effect of PROLI/NO on neointimal hyperplasia.

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7.  Tissue factor is rapidly induced in arterial smooth muscle after balloon injury.

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8.  Endogenous testosterone attenuates neointima formation after moderate coronary balloon injury in male swine.

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9.  Agonist-mediated tissue factor expression in cultured vascular smooth muscle cells. Role of Ca2+ mobilization and protein kinase C activation.

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10.  HO-1 and CO decrease platelet-derived growth factor-induced vascular smooth muscle cell migration via inhibition of Nox1.

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