Literature DB >> 24721237

The electrogenic cardiac sodium bicarbonate co-transporter (NBCe1) contributes to the reperfusion injury.

Juliana C Fantinelli1, Alejandro Orlowski2, Ernesto A Aiello1, Susana M Mosca3.   

Abstract

BACKGROUND: Although the participation of the electrogenic sodium/bicarbonate cotransporter (NBCe1) in the recovery from an intracellular acid load is recognized, its role in ischemia-reperfusion is still unclear. METHODS AND
RESULTS: Our objective was to assess the role of NBCe1 in reperfusion injury. We use selective functional antibodies against extracellular loop 3 (a-L3) and loop 4 (a-L4) of NBCe1. a-L3 inhibits and a-L4 stimulates NBCe1 activity. Isolated rat hearts were submitted to 40 min of coronary occlusion and 1 h of reperfusion. a-L3, a-L4 or S0859--selective Na(+)-HCO3(-) co-transport inhibitor--were administered during the initial 10 min of reperfusion. The infarct size (IS) was measured by triphenyltetrazolium chloride staining technique. Postischemic systolic and diastolic functions were also assessed. a-L3 and S0859 treatments decreased significantly (P < .05) the IS (16 ± 3% for a-L3 vs. 32 ± 5% in hearts treated with control nonimmune serum and 19 ± 3% for S0859 vs. 39 ± 2% in untreated hearts). Myocardial function during reperfusion improved after a-L3 treatment, but it was not modified by S0859. The infusion of a-L4 did not modify neither the IS nor myocardial function.
CONCLUSIONS: The NBCe1 hyperactivity during reperfusion leads to Na(+) and Ca(2+) loading, conducing to Ca(2+) overload and myocardial damage. Consistently, we have shown herein that the selective NBCe1 blockade with a-L3 exerted cardioprotection. This beneficial action strongly suggests that NBCe1 could be a potential target for the treatment of coronary disease.
Copyright © 2014 Elsevier Inc. All rights reserved.

Entities:  

Keywords:  Infarct size; Ischemia–Reperfusion; NBCe1; NBCe1 antibodies; S0859

Mesh:

Substances:

Year:  2014        PMID: 24721237     DOI: 10.1016/j.carpath.2014.03.003

Source DB:  PubMed          Journal:  Cardiovasc Pathol        ISSN: 1054-8807            Impact factor:   2.185


  6 in total

1.  The Na+/HCO3- co-transporter is protective during ischemia in astrocytes.

Authors:  Hang Yao; Priti Azad; Huiwen W Zhao; Juan Wang; Orit Poulsen; Beatriz C Freitas; Alysson R Muotri; Gabriel G Haddad
Journal:  Neuroscience       Date:  2016-10-04       Impact factor: 3.590

2.  The HVCN1 voltage-gated proton channel contributes to pH regulation in canine ventricular myocytes.

Authors:  Jianyong Ma; Xiaoqian Gao; Yutian Li; Thomas E DeCoursey; Gary E Shull; Hong-Sheng Wang
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Review 3.  Regulators of Slc4 bicarbonate transporter activity.

Authors:  Ian M Thornell; Mark O Bevensee
Journal:  Front Physiol       Date:  2015-06-12       Impact factor: 4.566

Review 4.  An overview of carbonic anhydrases and membrane channels of synoviocytes in inflamed joints.

Authors:  Min Jeong Ji; Jeong Hee Hong
Journal:  J Enzyme Inhib Med Chem       Date:  2019-12       Impact factor: 5.051

5.  NBCe1 Na+-HCO3- cotransporter ablation causes reduced apoptosis following cardiac ischemia-reperfusion injury in vivo.

Authors:  Kanimozhi Vairamani; Vikram Prasad; Yigang Wang; Wei Huang; Yinhua Chen; Mario Medvedovic; John N Lorenz; Gary E Shull
Journal:  World J Cardiol       Date:  2018-09-26

Review 6.  The Fundamental Role of Bicarbonate Transporters and Associated Carbonic Anhydrase Enzymes in Maintaining Ion and pH Homeostasis in Non-Secretory Organs.

Authors:  Dongun Lee; Jeong Hee Hong
Journal:  Int J Mol Sci       Date:  2020-01-04       Impact factor: 5.923

  6 in total

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