Literature DB >> 24702788

Diminished soluble levels of growth arrest specific protein 6 and tyrosine kinase receptor Axl in patients with rheumatoid arthritis.

Iman H Bassyouni1, Mohammed M El-Wakd1, Noha A Azab1, Rasha H Bassyouni2.   

Abstract

AIM: Growth arrest specific protein 6 (Gas-6) and its tyrosine kinase receptor Axl plays an important role in apoptosis, and regulation of innate immune response, therefore, we investigated their plasma concentrations in Rheumatoid arthritis (RA) patients and correlated them to clinical, laboratory and radiological parameters of the disease.
METHODS: Plasma from 77 RA patients and 50 normal healthy subjects were assayed for plasma Gas6 and Axl levels. Demographic, clinical and serological data were prospectively assessed. Rheumatoid arthritis disease activity was assessed using 28-joint Disease Activity Score (DAS-28) and functional capacity by modified health assessment questionnaire (mHAQ). Standardized x-rays for hands and feet were done to all participants.
RESULTS: The level of Gas6 and Axl were significantly decreased in the RA patients compared to those of the healthy control subjects. Levels of Gas6 correlated positively with Axl levels in both patients and healthy control. Gas6 levels were remarkably reduced in those patients with erosive RA than those without. Levels of Gas6 were found to be negatively correlated with the presence of erosive disease and positively correlated with DAS-28, ESR, Leucocytosis and IL6.
CONCLUSION: The plasma concentrations of Gas6 and Axl are altered in RA patients and thus may have a role in RA pathogenesis. Further mechanistic studies on the involvement of all TAM receptors tyrosine kinases pathway in RA are needed to help in understanding the pathogenesis and possibly aid in diagnosis and future treatments of RA especially for patients with erosive disease.
© 2014 Asia Pacific League of Associations for Rheumatology and Wiley Publishing Asia Pty Ltd.

Entities:  

Keywords:  Axl; apoptosis; growth arrest-specific protein 6; inflammation; receptor tyrosine kinase; rheumatoid arthritis

Mesh:

Substances:

Year:  2014        PMID: 24702788     DOI: 10.1111/1756-185X.12367

Source DB:  PubMed          Journal:  Int J Rheum Dis        ISSN: 1756-1841            Impact factor:   2.454


  9 in total

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Review 4.  New Insights into the Role of Tyro3, Axl, and Mer Receptors in Rheumatoid Arthritis.

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Review 5.  Critical Role of Synovial Tissue-Resident Macrophage and Fibroblast Subsets in the Persistence of Joint Inflammation.

Authors:  Samuel Kemble; Adam P Croft
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6.  AXL Inhibits Proinflammatory Factors to Relieve Rheumatoid Arthritis Pain by Regulating the TLR4/NF-κB Signaling Pathway.

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7.  Growth Arrest-Specific 6 Protein in Patients with Sjögren Syndrome: Determination of the Plasma Level and Expression in the Labial Salivary Gland.

Authors:  Chen-Hung Chen; Hsiang-Cheng Chen; Chi-Ching Chang; Yi-Jen Peng; Chien-Hsing Lee; Yi-Shing Shieh; Yi-Jen Hung; Yuh-Feng Lin
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8.  Analysis of receptor tyrosine kinase genetics identifies two novel risk loci in GAS6 and PROS1 in Behçet's disease.

Authors:  Jieying Qin; Lin Li; Donglei Zhang; Hongsong Yu; Handan Tan; Jun Zhang; Bolin Deng; Aize Kijlstra; Peizeng Yang
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9.  Tyro3/Axl/Mertk-deficient mice develop bone marrow edema which is an early pathological marker in rheumatoid arthritis.

Authors:  Claire E J Waterborg; Marije I Koenders; Peter L E M van Lent; Peter M van der Kraan; Fons A J van de Loo
Journal:  PLoS One       Date:  2018-10-18       Impact factor: 3.240

  9 in total

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