| Literature DB >> 24686393 |
Stefan Deneberg1, Meena Kanduri2, Dina Ali1, Sofia Bengtzen1, Mohsen Karimi1, Ying Qu1, Eva Kimby1, Larry Mansouri3, Richard Rosenquist3, Andreas Lennartsson4, Sören Lehmann1.
Abstract
A commonly deleted region in chronic lymphocytic leukemia (CLL) is the 11q22-23 region, which encompasses the ATM gene. Evidence suggests that tumor suppressor genes other than ATM are likely to be involved in CLL with del(11q). A microRNA (miR) cluster including the miR-34b and miR-34c genes is located, among other genes, within the commonly deleted region (CDR) at 11q. Interestingly, these miRs are part of the TP53 network and have been shown to be epigenetically regulated. In this study, we investigated the expression and methylation status of these miRs in a well-characterized cohort of CLL, including cases with/without 11q-deletion. We show that the miR-34b/c promoter was aberrantly hypermethylated in a large proportion of CLL cases (48%, 25/52 cases). miR-34b/c expression correlated inversely to DNA methylation (P = 0.003), and presence of high H3K37me3 further suppressed expression regardless of methylation status. Furthermore, increased miR-34b/c methylation inversely correlated with the presence of 11q-deletion, indicating that methylation and del(11q) independently silence these miRs. Finally, 5-azacytidine and trichostatin A exposure synergistically increased the expression of miR-34b/c in CLL cells, and transfection of miR-34b or miR-34c into HG3 CLL cells significantly increased apoptosis. Altogether, our novel data suggest that miR-34b/c is a candidate tumor suppressor that is epigenetically silenced in CLL.Entities:
Keywords: DNA methylation; chronic lymphatic leukemia; epigenetics; micro-RNA
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Year: 2014 PMID: 24686393 PMCID: PMC4053441 DOI: 10.4161/epi.28603
Source DB: PubMed Journal: Epigenetics ISSN: 1559-2294 Impact factor: 4.528