Literature DB >> 24683216

Effects of zileuton on the development of autoimmune myocarditis in an experimental rat model.

Ender Odemis1, Sadi Turkay1, Cemile Koca2, Halil Kiyici3, Ahmet Karadag4, Reyhan Bayrak5, Emin Mete4, Ferhat Catal4, Ramazan Yigitoglu2.   

Abstract

BACKGROUND: Myocarditis is associated with high morbidity and mortality in childhood, but the pathogenesis of this disease is still unclear. Current knowledge indicates that complex immunopathogenic mechanisms are involved. It is understood that leukotrienes play an important role in the inflammation associated with asthma, and recent reports indicate that leukotrienes participate in immune processes and in autoimmunity.
OBJECTIVE: The aim of this study was to assess the role of leukotriene synthesis in the development of myocardial inflammation and necrosis during myocarditis.
METHODS: The effect of zileuton, a leukotriene synthesis inhibitor, was assessed in an experimental model of autoimmune myocarditis in rats. Healthy adult (10-week-old) male Wistar albino rats were randomly divided into 3 groups. Groups A and B received injections of 1.0 mg porcine cardiac myosin to induce autoimmune myocarditis and group C (the control group) received phosphate-buffered saline. Group B also received zileuton by oral gavage at 100 mg/kg · d(-1). Myocardial inflammation was assessed biochemically via serum concentrations of creatine kinase MB subunit (CK-MB) and troponin T. Cardiac tissue was assessed macroscopically (0 = no inflammation; 1 = a small discolored focus; 2 = diffuse discolored areas covering less than half of the cardiac surface; 3 = diffuse discolored areas covering more than half of the cardiac surface) and microscopically (0 = no inflammation; 1 = ⪯5% infiltration; 2 = 5% to <10% infiltration; 3 = 10% to < 20% infiltration; 4 = >20% infiltration).
RESULTS: Twenty-four rats were divided equally into 3 groups. All rats survived the duration of the study. After 21 days, all rats were euthanized. No significant differences were found between groups A and B in terms of serum concentrations of CK-MB or troponin T. The microscopic pathology score was significantly lower in group B (myosin + zileuton) than in group A (myosin only) (0.12 [0.35] vs 1.25 [1.03]; P = 0.023). The microscopic pathology score was significantly higher in group A than in group C (1.25 [1.03] vs 0; P < 0.01), but the difference between groups B and C was not statistically significant. The macroscopic pathology score was significantly higher in group A than in group B (1.37 [0.91] vs 0.37 [0.51]; P = 0.029). The macroscopic pathology score was significantly higher in group A than in group C (1.37 [0.91] vs 0; P < 0.01). There was no significant difference in macroscopic scores between groups B and C (0.37 [0.51] vs 0).
CONCLUSIONS: In this study of the effects of zileuton in an experimental rat model of autoimmune myocarditis, no significant differences were found in the serum concentrations of the biochemical markers in the 2 groups administered myosin. However, lower macroscopic and microscopic inflammation scores in the zileuton-treated group (group B) compared with the group administered only myosin (group A) suggest that zileuton, a leukotriene-synthesis inhibitor, may suppress the development of experimental autoimmune myocarditis in rats.

Entities:  

Keywords:  autoimmunity; leukotrienes; myocarditis; rats; zileuton

Year:  2007        PMID: 24683216      PMCID: PMC3967347          DOI: 10.1016/j.curtheres.2007.08.002

Source DB:  PubMed          Journal:  Curr Ther Res Clin Exp        ISSN: 0011-393X


  16 in total

Review 1.  Interactions between leukotrienes and other inflammatory mediators/modulators in the microvasculature.

Authors:  P Hedqvist; N Gautam; L Lindbom
Journal:  Am J Respir Crit Care Med       Date:  2000-02       Impact factor: 21.405

Review 2.  Leukotrienes, sphingolipids, and leukocyte trafficking.

Authors:  Adam C Yopp; Gwendolyn J Randolph; Jonathan S Bromberg
Journal:  J Immunol       Date:  2003-07-01       Impact factor: 5.422

Review 3.  The role of leukotrienes in the pathophysiology of inflammatory disorders: is there a case for revisiting leukotrienes as therapeutic targets?

Authors:  J N Sharma; L A Mohammed
Journal:  Inflammopharmacology       Date:  2006-03       Impact factor: 4.473

4.  Suppression of experimental autoimmune myocarditis by sodium fusidate (fusidin).

Authors:  M Milenković; D Vucićević; P Milosavljević; N Arsenović Ranin; Z Stojić Vukanić; M Colić
Journal:  Pharmacol Res       Date:  2005-10-07       Impact factor: 7.658

Review 5.  Autoimmunity in myocarditis: relevance of animal models.

Authors:  S A Huber
Journal:  Clin Immunol Immunopathol       Date:  1997-05

6.  A novel experimental model of giant cell myocarditis induced in rats by immunization with cardiac myosin fraction.

Authors:  M Kodama; Y Matsumoto; M Fujiwara; F Masani; T Izumi; A Shibata
Journal:  Clin Immunol Immunopathol       Date:  1990-11

7.  Cardiac troponin T in patients with clinically suspected myocarditis.

Authors:  B Lauer; C Niederau; U Kühl; M Schannwell; M Pauschinger; B E Strauer; H P Schultheiss
Journal:  J Am Coll Cardiol       Date:  1997-11-01       Impact factor: 24.094

Review 8.  Leukotrienes: underappreciated mediators of innate immune responses.

Authors:  Marc Peters-Golden; Claudio Canetti; Peter Mancuso; Michael J Coffey
Journal:  J Immunol       Date:  2005-01-15       Impact factor: 5.422

Review 9.  Myocarditis: the immunologist's view on pathogenesis and treatment.

Authors:  Julia Burian; Peter Buser; Urs Eriksson
Journal:  Swiss Med Wkly       Date:  2005-06-25       Impact factor: 2.193

10.  Coxsackievirus B-3 myocarditis in Balb/c mice. Evidence for autoimmunity to myocyte antigens.

Authors:  S A Huber; P A Lodge
Journal:  Am J Pathol       Date:  1984-07       Impact factor: 4.307

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