Literature DB >> 24668949

Dexrazoxane exposure and risk of secondary acute myeloid leukemia in pediatric oncology patients.

Alix E Seif1, Dana M Walker, Yimei Li, Yuan-Shung V Huang, Marko Kavcic, Kari Torp, Rochelle Bagatell, Brian T Fisher, Richard Aplenc.   

Abstract

BACKGROUND: Dexrazoxane may reduce anthracycline-associated cardiotoxicity in pediatric cancer patients. However, concerns of secondary acute myeloid leukemia (AML) have led to restrictions on pediatric dexrazoxane use in Europe. Published data about dexrazoxane-associated secondary AML are limited and conflicting. We sought to estimate the secondary AML risk in children receiving dexrazoxane after anthracycline exposure. PROCEDURE: A retrospective cohort of children with newly identified malignancies (excluding AML) receiving anthracyclines between January 1, 1999 and March 31, 2011 was established using the Pediatric Health Information System (PHIS). Patients were followed for all subsequent admissions to identify dexrazoxane exposures and secondary AML, defined by AML ICD-9 codes and AML induction chemotherapy. Logistic regression was used to model the association of dexrazoxane and secondary AML risk. A propensity score was used to adjust for measurable confounding.
RESULTS: Of 15,532 patients in the cohort exposed to anthracyclines, 1,406 received dexrazoxane. The secondary AML rate was 0.21% (3 of 1,046) in dexrazoxane-exposed and 0.55% (77 of 14,126) in unexposed patients. In a propensity score-adjusted multivariate analysis, dexrazoxane exposure was not associated with an increased risk of secondary AML, OR = 0.38, 95% CI 0.11-1.26.
CONCLUSIONS: Dexrazoxane was not associated with an increased risk of secondary AML in a large cohort of pediatric cancer patients receiving anthracyclines in US hospitals. While these data support dexrazoxane's safety in the general pediatric oncology population, additional studies are needed to confirm these findings and to quantify dexrazoxane's long-term cardioprotective effects.
© 2014 Wiley Periodicals, Inc.

Entities:  

Keywords:  cardiotoxicity after cancer therapy; dexrazoxane; epidemiology; secondary malignancy

Mesh:

Substances:

Year:  2014        PMID: 24668949      PMCID: PMC4177031          DOI: 10.1002/pbc.25043

Source DB:  PubMed          Journal:  Pediatr Blood Cancer        ISSN: 1545-5009            Impact factor:   3.167


  25 in total

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Journal:  Eur J Cancer       Date:  2006-09-20       Impact factor: 9.162

Review 4.  Dexrazoxane : a review of its use for cardioprotection during anthracycline chemotherapy.

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Journal:  Drugs       Date:  2005       Impact factor: 9.546

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10.  Long-term results of the pediatric oncology group studies for childhood acute lymphoblastic leukemia 1984-2001: a report from the children's oncology group.

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Journal:  Leukemia       Date:  2009-12-17       Impact factor: 11.528

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8.  Decreased Soluble Guanylate Cyclase Contributes to Cardiac Dysfunction Induced by Chronic Doxorubicin Treatment in Mice.

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Journal:  Antioxid Redox Signal       Date:  2016-09-08       Impact factor: 8.401

Review 9.  Research progress of cardioprotective agents for prevention of anthracycline cardiotoxicity.

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Review 10.  The beneficial effects of reducing NLRP3 inflammasome activation in the cardiotoxicity and the anti-cancer effects of doxorubicin.

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