Literature DB >> 24639367

Aerobic training in patients with anoctamin 5 myopathy and hyperckemia.

Christoffer R Vissing1, Nicolai Preisler, Edith Husu, Kira P Prahm, John Vissing.   

Abstract

INTRODUCTION: Anoctamin 5 deficiency has recently been defined to cause limb-girdle muscular dystrophy type 2L (LGMD2L) with pronounced hyperCKemia. No treatment interventions have been made so far in this condition.
METHODS: In 6 patients with LGMD2L, we studied the effect of home-based, pulse-watch monitored, moderate-intensity exercise on a cycle ergometer for 30 minutes, 3 times weekly, for 10 weeks. Plasma creatine kinase (CK) was assessed before, during, and after the program as a marker of muscle damage. Primary outcome measures were maximum oxygen uptake (VO(2max)) and time in the 5-repetitions-sit-to-stand test (FRSTST).
RESULTS: Training resulted in improvements in VO(2max) (27 ± 7%; P = 0.0001) and FRSTST time (35 ± 12%; P = 0.007). Improvements in physiologic and functional muscle testing were accompanied by stable CK levels and no reports of adverse effects.
CONCLUSIONS: These findings suggest that supervised aerobic exercise training is safe and effective in improving oxidative capacity and muscle function in patients with anoctamin 5 deficiency.
Copyright © 2013 Wiley Periodicals, Inc.

Entities:  

Keywords:  ANO-5; aerobic training; anoctamin 5; limb-girdle muscular dystrophy type 2L; neuromuscular disorders

Mesh:

Substances:

Year:  2014        PMID: 24639367     DOI: 10.1002/mus.24112

Source DB:  PubMed          Journal:  Muscle Nerve        ISSN: 0148-639X            Impact factor:   3.217


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