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24 in total

1. Rapid loss of superoxide dismutase activity during antigen-induced asthmatic response.

Authors: S A Comhair; P R Bhathena; R A Dweik; M Kavuru; S C Erzurum
Journal: Lancet Date: 2000-02-19 Impact factor: 79.321

Review 2. Redox environment of the cell as viewed through the redox state of the glutathione disulfide/glutathione couple.

Authors: F Q Schafer; G R Buettner
Journal: Free Radic Biol Med Date: 2001-06-01 Impact factor: 7.376

3. Tc-99m-HMPAO uptake by bronchoalveolar cells.

Authors: Hatice Durak; Oğuz Kilinç; Türkan Ertay; Eyüp Sabri Uçan; Aydanur Kargi; Gamze Capa Kaya; Banu Sis
Journal: Ann Nucl Med Date: 2003-04 Impact factor: 2.668

4. Eosinophils are a major source of nitric oxide-derived oxidants in severe asthma: characterization of pathways available to eosinophils for generating reactive nitrogen species.

Authors: J C MacPherson; S A Comhair; S C Erzurum; D F Klein; M F Lipscomb; M S Kavuru; M K Samoszuk; S L Hazen
Journal: J Immunol Date: 2001-05-01 Impact factor: 5.422

5. Increased airway inflammation with segmental versus aerosol antigen challenge.

Authors: W J Calhoun; N N Jarjour; G J Gleich; C A Stevens; W W Busse
Journal: Am Rev Respir Dis Date: 1993-06

6. NO chemical events in the human airway during the immediate and late antigen-induced asthmatic response.

Authors: R A Dweik; S A Comhair; B Gaston; F B Thunnissen; C Farver; M J Thomassen; M Kavuru; J Hammel; H M Abu-Soud; S C Erzurum
Journal: Proc Natl Acad Sci U S A Date: 2001-02-20 Impact factor: 11.205

7. Extracellular glutathione peroxidase induction in asthmatic lungs: evidence for redox regulation of expression in human airway epithelial cells.

Authors: S A Comhair; P R Bhathena; C Farver; F B Thunnissen; S C Erzurum
Journal: FASEB J Date: 2001-01 Impact factor: 5.191

Redox imaging of inflammation in asthma.

1. Rapid loss of superoxide dismutase activity during antigen-induced asthmatic response.

Review 2. Redox environment of the cell as viewed through the redox state of the glutathione disulfide/glutathione couple.

3. Tc-99m-HMPAO uptake by bronchoalveolar cells.

4. Eosinophils are a major source of nitric oxide-derived oxidants in severe asthma: characterization of pathways available to eosinophils for generating reactive nitrogen species.

5. Increased airway inflammation with segmental versus aerosol antigen challenge.

6. NO chemical events in the human airway during the immediate and late antigen-induced asthmatic response.

7. Extracellular glutathione peroxidase induction in asthmatic lungs: evidence for redox regulation of expression in human airway epithelial cells.

8. Eosinophils generate brominating oxidants in allergen-induced asthma.

9. Increase in endothelial cell glutathione and precursor amino acid uptake by diethyl maleate and hyperoxia.

10. 99Tcm-labelled meso-HMPAO and glutathione content of human lung tumours.

Review 1. New Insights in Oxidant Biology in Asthma.